RETRACTION: LINC02381 Aggravates Breast Cancer Through the miR-1271-5p/FN1 Axis to Activate PI3K/AKT Pathway.

IF 3 2区 医学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
Molecular Carcinogenesis Pub Date : 2025-02-01 Epub Date: 2024-12-16 DOI:10.1002/mc.23865
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引用次数: 0

Abstract

Retraction: S. Huang, P. Huang, H. Wu, S. Wang, and G. Liu, "LINC02381 Aggravates Breast Cancer Through the miR-1271-5p/FN1miR-1271-5p/FN1 Axis to Activate PI3KPI3K/AKT/AKT Pathway," Molecular Carcinogenesis 61, no. 3 (2022): 346-358, https://doi.org/10.1002/mc.23375. The above article, published online on 9 December 2021 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors and Wiley Periodicals LLC. The retraction has been agreed due to the authors' discovery that the results could not be replicated under the same conditions. The authors' repeated experiments were not completely consistent with the previous results published in this article, which directly affects the conclusions of the article. Therefore, the parties agree that the article must be retracted.

回归:LINC02381 通过 miR-1271-5p/FN1 轴激活 PI3K/AKT 通路,加重乳腺癌病情
撤回:S. Huang, P. Huang, H. Wu, S. Wang, and G. Liu, "LINC02381 Aggravates Breast Cancer Through the miR-1271-5p/FN1miR-1271-5p/FN1 Axis to Activate PI3KPI3K/AKT/AKT Pathway," Molecular Carcinogenesis 61, no.3 (2022): 346-358, https://doi.org/10.1002/mc.23375。上述文章于 2021 年 12 月 9 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经作者与 Wiley Periodicals LLC 协议,该文章已被撤回。同意撤稿的原因是作者发现在相同条件下无法重复实验结果。作者的重复实验结果与本文之前发表的结果不完全一致,这直接影响了文章的结论。因此,双方同意必须撤回该文章。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Molecular Carcinogenesis
Molecular Carcinogenesis 医学-生化与分子生物学
CiteScore
7.30
自引率
2.20%
发文量
112
审稿时长
2 months
期刊介绍: Molecular Carcinogenesis publishes articles describing discoveries in basic and clinical science of the mechanisms involved in chemical-, environmental-, physical (e.g., radiation, trauma)-, infection and inflammation-associated cancer development, basic mechanisms of cancer prevention and therapy, the function of oncogenes and tumors suppressors, and the role of biomarkers for cancer risk prediction, molecular diagnosis and prognosis.
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