Cell-type-specific splicing of transcription regulators and Ptbp1 by Rbfox1/2/3 in the developing neocortex

Xiangbin Ruan, Kaining Hu, Yalan Yang, Runwei Yang, Elizabeth Tseng, Bowei Kang, Aileen Kauffman, Rong Zhong, Xiaochang Zhang
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Abstract

How master splicing regulators crosstalk with each other and to what extent transcription regulators are differentially spliced remain unclear in the developing brain. Here, cell-type-specific RNA-Seq of the developing neocortex uncover that transcription regulators are enriched for differential splicing, altering protein isoforms or inducing nonsense-mediated mRNA decay. Transient expression of Rbfox proteins in radial glia progenitors induces neuronal splicing events preferentially in transcription regulators such as Meis2 and Tead1. Surprisingly, Rbfox proteins promote the inclusion of a mammal-specific alternative exon and a previously undescribed poison exon in Ptbp1. Simultaneous ablation of Rbfox1/2/3 in the neocortex downregulates neuronal isoforms and disrupts radial neuronal migration. Furthermore, the progenitor isoform of Meis2 promotes Tgfb3 transcription, while the Meis2 neuron isoform promotes neuronal differentiation. These observations indicate that transcription regulators are differentially spliced between cell types in the developing neocortex.
在发育中的新皮层中,Rbfox1/2/3对转录调节因子和Ptbp1进行细胞类型特异性剪接
在发育中的大脑中,主剪接调控因子如何相互串联以及转录调控因子在多大程度上被差异剪接仍不清楚。在这里,对发育中的新皮质进行的细胞类型特异性 RNA-Seq 研究发现,转录调节因子富集于差异剪接,改变蛋白质同工型或诱导无义介导的 mRNA 衰减。在径向胶质祖细胞中瞬时表达Rbfox蛋白会诱导神经元剪接事件,而Meis2和Tead1等转录调控因子则是首选。令人惊讶的是,Rbfox 蛋白促进了哺乳动物特异性替代外显子和以前未描述过的 Ptbp1 毒素外显子的包含。同时消减新皮质中的Rbfox1/2/3会下调神经元同工酶,并破坏神经元的径向迁移。此外,Meis2的祖细胞同工酶促进Tgfb3的转录,而Meis2的神经元同工酶则促进神经元的分化。这些观察结果表明,转录调节因子在发育中的新皮层细胞类型之间存在不同的剪接。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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