Histone β-hydroxybutyrylation is critical in reversal of sarcopenia.

IF 8 1区 医学 Q1 CELL BIOLOGY
Aging Cell Pub Date : 2024-07-30 DOI:10.1111/acel.14284
Qiquan Wang, Xinqiang Lan, Hao Ke, Siman Xu, Chunping Huang, Jiali Wang, Xiang Wang, Tiane Huang, Xia Wu, Mengxin Chen, Yingqi Guo, Lin Zeng, Xiao-Li Tian, Yang Xiang
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Abstract

Sarcopenia, a leading cause for global disability and mortality, is an age-related muscular disorder, characterized by accelerated muscle mass loss and functional decline. It is known that caloric restriction (CR), ketogenic diet or endurance exercise lessen sarcopenia and elevate circulating β-hydroxybutyrate (β-HB) levels. Whether the elevated β-HB is essential to the reversal of sarcopenia, however, remains unclear. Here we show in both Caenorhabditis elegans and mouse models that an increase of β-HB reverse myofiber atrophy and improves motor functions at advanced ages. β-HB-induced histone lysine β-hydroxybutyrylation (Kbhb) is indispensable for the reversal of sarcopenia. Histone Kbhb enhances transcription of genes associated with mitochondrial pathways, including oxidative phosphorylation, ATP metabolic process and aerobic respiration. This ultimately leads to improve mitochondrial integrity and enhance mitochondrial respiration. The histone Kbhb are validated in mouse model with CR. Thus, we demonstrate that β-HB induces histone Kbhb, increases mitochondrial function, and reverses sarcopenia.

Abstract Image

组蛋白β-羟基丁酰化在逆转肌肉疏松症中至关重要。
肌肉疏松症是导致全球残疾和死亡的一个主要原因,是一种与年龄有关的肌肉疾病,其特点是肌肉质量加速流失和功能衰退。众所周知,热量限制(CR)、生酮饮食或耐力锻炼可减轻肌肉疏松症,并提高循环中的β-羟丁酸(β-HB)水平。然而,β-HB 的升高是否是逆转肌肉疏松症的关键,目前仍不清楚。在这里,我们在秀丽隐杆线虫和小鼠模型中发现,β-羟丁酸的增加可逆转肌纤维萎缩,并改善高龄时的运动功能。β-HB诱导的组蛋白赖氨酸β-羟基丁酰化(Kbhb)是逆转肌肉疏松不可或缺的因素。组蛋白 Kbhb 可增强与线粒体途径相关的基因转录,包括氧化磷酸化、ATP 代谢过程和有氧呼吸。这最终会改善线粒体的完整性,增强线粒体呼吸。组蛋白 Kbhb 在 CR 小鼠模型中得到了验证。因此,我们证明了 β-HB 可诱导组蛋白 Kbhb、增强线粒体功能并逆转肌肉疏松症。
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来源期刊
Aging Cell
Aging Cell Biochemistry, Genetics and Molecular Biology-Cell Biology
自引率
2.60%
发文量
212
期刊介绍: Aging Cell is an Open Access journal that focuses on the core aspects of the biology of aging, encompassing the entire spectrum of geroscience. The journal's content is dedicated to publishing research that uncovers the mechanisms behind the aging process and explores the connections between aging and various age-related diseases. This journal aims to provide a comprehensive understanding of the biological underpinnings of aging and its implications for human health. The journal is widely recognized and its content is abstracted and indexed by numerous databases and services, which facilitates its accessibility and impact in the scientific community. These include: Academic Search (EBSCO Publishing) Academic Search Alumni Edition (EBSCO Publishing) Academic Search Premier (EBSCO Publishing) Biological Science Database (ProQuest) CAS: Chemical Abstracts Service (ACS) Embase (Elsevier) InfoTrac (GALE Cengage) Ingenta Select ISI Alerting Services Journal Citation Reports/Science Edition (Clarivate Analytics) MEDLINE/PubMed (NLM) Natural Science Collection (ProQuest) PubMed Dietary Supplement Subset (NLM) Science Citation Index Expanded (Clarivate Analytics) SciTech Premium Collection (ProQuest) Web of Science (Clarivate Analytics) Being indexed in these databases ensures that the research published in Aging Cell is discoverable by researchers, clinicians, and other professionals interested in the field of aging and its associated health issues. This broad coverage helps to disseminate the journal's findings and contributes to the advancement of knowledge in geroscience.
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