[Transient neurological deficits in a patient with chronic subdural hematoma].

Q4 Medicine
Clinical Neurology Pub Date : 2024-06-27 Epub Date: 2024-06-20 DOI:10.5692/clinicalneurol.cn-001975
Misa Ishiuchi, Yuichiro Inatomi, Rihito Yamamura, Makoto Nakajima, Toshiro Yonehara
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引用次数: 0

Abstract

A 62-year-old, right-handed man was diagnosed with asymptomatic bilateral chronic subdural hematomas and underwent hematoma removal on the left side only. At 1 month after surgery, he was admitted to our hospital because he began to have one or two attacks/day of apraxia of speech and dysesthesia of the right hand with a duration of approximately 5 ‍min. The left hematoma had not re-expanded, but fluid-attenuated inversion resonance imaging showed hyperintense lesions in the sulci adjacent to the hematoma. Moreover, single-photon emission computed tomography revealed low-uptake lesions in the left cerebrum adjacent to the hematoma. Electroencephalogram showed no abnormalities, and CT angiography showed a slight deviation of the left middle cerebral arteries due to the hematoma. The attacks disappeared within 10 days, although the volume of the hematoma was unchanged. It was suggested that his transient neurological deficits were caused by cerebral ischemia related to chronic subdural hematoma.

[慢性硬膜下血肿患者短暂的神经功能缺损]。
一名 62 岁的右撇子男子被诊断为无症状双侧慢性硬膜下血肿,仅接受了左侧血肿清除手术。术后 1 个月,他开始每天发作一到两次语言障碍和右手感觉障碍,持续时间约为 5 ‍分钟,因此被送入我院。左侧血肿没有再扩大,但体液减弱反转共振成像显示血肿附近的沟内有高强度病变。此外,单光子发射计算机断层扫描显示,血肿附近的左侧大脑有低摄取病变。脑电图未显示异常,CT 血管造影显示左侧大脑中动脉因血肿而轻微偏离。虽然血肿的体积没有变化,但发作在 10 天内消失了。有人认为,他的一过性神经功能缺损是由与慢性硬膜下血肿有关的脑缺血引起的。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Clinical Neurology
Clinical Neurology Medicine-Neurology (clinical)
CiteScore
0.30
自引率
0.00%
发文量
147
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