The role of IL-1B in breast cancer bone metastasis

IF 3.4 2区 医学 Q2 Medicine
Jiabao Zhou, Penelope D. Ottewell
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引用次数: 0

Abstract

Interleukin-1B (IL-1B) is a potent pro-inflammatory cytokine that plays multiple, pivotal roles, in the complex interplay between breast cancer cells and the bone microenvironment. IL-1B is involved in the growth of the primary tumours, regulation of inflammation within the tumour microenvironment, promotion of epithelial to mesenchymal transition (EMT), migration and invasion. Moreover, when breast cancer cells arrive in the bone microenvironment there is an upregulation of IL-1B which promotes the creation of a conducive niche for metastatic breast cancer cells as well as stimulating initiation of the vicious cycle of bone metastasis. Pre-clinical studies have demonstrated that inhibition of IL-1 signalling reduces bone metastasis from oestrogen receptor positive/triple-negative breast cancers in various mouse models. However, effects on primary tumours and soft tissue metastasis remain controversial with some studies showing increased tumour growth in these sites, whilst others show no effects. Notably, combining anti-IL-1 therapy with standard-of-care treatments, such as chemotherapy and immunotherapy, has been demonstrated to reduce the growth of primary tumours, bone metastasis, as well as metastatic outgrowth in other organs. This review focuses on the mechanisms by which IL-1B promotes breast cancer bone metastasis.

IL-1B 在乳腺癌骨转移中的作用
白细胞介素-1B(IL-1B)是一种强效促炎细胞因子,在乳腺癌细胞与骨微环境的复杂相互作用中发挥着多重关键作用。IL-1B 参与原发性肿瘤的生长、肿瘤微环境中炎症的调节、促进上皮到间质的转化(EMT)、迁移和侵袭。此外,当乳腺癌细胞进入骨微环境时,IL-1B 会上调,从而为转移性乳腺癌细胞创造有利的龛位,并刺激骨转移恶性循环的开始。临床前研究表明,在各种小鼠模型中,抑制 IL-1 信号可减少雌激素受体阳性/三阴性乳腺癌的骨转移。然而,对原发肿瘤和软组织转移的影响仍存在争议,一些研究显示这些部位的肿瘤生长增加,而另一些研究则显示没有影响。值得注意的是,将抗IL-1疗法与化疗和免疫疗法等标准疗法相结合,已被证明可减少原发性肿瘤的生长、骨转移以及其他器官的转移生长。本综述重点探讨IL-1B促进乳腺癌骨转移的机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
7.20
自引率
2.90%
发文量
50
审稿时长
34 days
期刊介绍: The Journal of Bone Oncology is a peer-reviewed international journal aimed at presenting basic, translational and clinical high-quality research related to bone and cancer. As the first journal dedicated to cancer induced bone diseases, JBO welcomes original research articles, review articles, editorials and opinion pieces. Case reports will only be considered in exceptional circumstances and only when accompanied by a comprehensive review of the subject. The areas covered by the journal include: Bone metastases (pathophysiology, epidemiology, diagnostics, clinical features, prevention, treatment) Preclinical models of metastasis Bone microenvironment in cancer (stem cell, bone cell and cancer interactions) Bone targeted therapy (pharmacology, therapeutic targets, drug development, clinical trials, side-effects, outcome research, health economics) Cancer treatment induced bone loss (epidemiology, pathophysiology, prevention and management) Bone imaging (clinical and animal, skeletal interventional radiology) Bone biomarkers (clinical and translational applications) Radiotherapy and radio-isotopes Skeletal complications Bone pain (mechanisms and management) Orthopaedic cancer surgery Primary bone tumours Clinical guidelines Multidisciplinary care Keywords: bisphosphonate, bone, breast cancer, cancer, CTIBL, denosumab, metastasis, myeloma, osteoblast, osteoclast, osteooncology, osteo-oncology, prostate cancer, skeleton, tumour.
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