{"title":"Sindrom Wellens Tipe B dengan Penyumbatan pada Satu Pembuluh Darah Koroner: Laporan Kasus","authors":"Edmond L. Jim","doi":"10.35790/msj.v6i1.52494","DOIUrl":null,"url":null,"abstract":"Abstract: Wellens syndrome describes a pattern of electrocardiographic (ECG) changes, especially typical T waves, that threaten the patient's safety if further examination and management are delayed. We reported a 52-year-old male with a history of previous heart attacks. The patient had a history of hypertension, hypercholestrolemia, and active smoker. Vital signs indicated stable hemodynamics. On ECG examination, a deep inversion T wave was obtained in the V2-V6 leads. In echocardio-graphy, normal heart chamber dimensions, concentric LVH, with grade I diastolic dysfunction, LV EF 52% (SIMPSONS BP), apical to mid-anterior hypokinetics, other normokinetic segments, MR Mild, with normal RV contractility. Patient was carried out elective percutaneous coronary intervention (PCI) with the results of one kulprit lesion found in LAD, 50-60% proximal-mid stenosis, 80% distal tubular stenosis, discrete stenosis 50-60% D2, and Promus Premiere 2.5x20 mm stent installation in the distal LAD. The result of TIMI flow 3 angiography evaluation was no dissection or residual stenosis that concluded CAD 1VD post DCA-PCI with 1 Stent in LAD. After the PCI, complaint of chest pain was absent, vital sign examination and physical examinations were within normal limits. The therapy provided consisted of 0.9% NaCl infusion 500cc/24 hours intravenously for hydration after PCI, and dual antiplatelets After day-3 treatment there was no increase in urea and creatinine; therefore, outpatient treatment was carried out followed by phase 2 cardiac rehabilitation.\nKeywords: Wellens’ syndrome; typical T wave; stenosis; left anterior descending artery\n \nAbstrak: Sindrom Wellens menggambarkan pola perubahan elektrokardiografi (EKG), terutama gelombang T tipikal yang mengancam keselamatan pasien bila terlambat dilakukan pemeriksaan dan penatalaksanaan lebih lanjut. Kami melaporkan seorang laki-laki beruria 52 tahun dengan riwayat serangan jantung sebelumnya. Pasien memiliki riwayat hipertensi, riwayat hiperkolestrolemia, dan perokok aktif. Tanda vital menunjukkan hemodinamik stabil. Pada pemeriksaan EKG didapatkan gelombang T inversi dalam pada sadapan V2-V6. Pada ekokardiografi didapatkan dimensi ruang jantung normal, LVH konsentrik, dengan disfungsi diastolik grade I, LV EF 52% (SIMPSONS BP), hipokinetik apikal hingga mid anterior, segmen lain normokinetik, MR Mild , dengan kontraktilitas RV normal. Pasien dilakukan intervensi koroner perkutan elektif dengan hasil satu lesi kulprit terdapat pada LAD, stenosis 50-60% proksimal-mid, stenosis tubular 80% distal, stenosis diskret 50-60% D2, dan dilakukan pemasangan stent Promus Premiere 2,5x20mm di distal LAD. Hasil evaluasi angiografi TIMI flow 3, tidak terdapat diseksi, ataupun stenosis residual yang menyimpulkan CAD 1VD post DCA-PCI dengan 1 Stent di LAD. Pasca tindakan, keluhan nyeri dada tidak ada, pemeriksaan tanda vital dan pemeriksaan fisik dalam batas normal. Terapi yang diberikan terdiri dari infus NaCl 0,9% 500cc/24jam intravena untuk hidrasi setelah IKP dan dual antiplatelet. Setelah perawatan hari ke 3 tidak didapatkan peningkatan ureum dan kreatinin, dilakukan rawat jalan yang dilanjutkan dengan rehabilitasi jantung fase 2.\nKata kunci: sindrom Wellens; gelombang T tipikal; stenosis; left anterior descending artery","PeriodicalId":118600,"journal":{"name":"Medical Scope Journal","volume":"3 4","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2023-12-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Medical Scope Journal","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.35790/msj.v6i1.52494","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
Abstract: Wellens syndrome describes a pattern of electrocardiographic (ECG) changes, especially typical T waves, that threaten the patient's safety if further examination and management are delayed. We reported a 52-year-old male with a history of previous heart attacks. The patient had a history of hypertension, hypercholestrolemia, and active smoker. Vital signs indicated stable hemodynamics. On ECG examination, a deep inversion T wave was obtained in the V2-V6 leads. In echocardio-graphy, normal heart chamber dimensions, concentric LVH, with grade I diastolic dysfunction, LV EF 52% (SIMPSONS BP), apical to mid-anterior hypokinetics, other normokinetic segments, MR Mild, with normal RV contractility. Patient was carried out elective percutaneous coronary intervention (PCI) with the results of one kulprit lesion found in LAD, 50-60% proximal-mid stenosis, 80% distal tubular stenosis, discrete stenosis 50-60% D2, and Promus Premiere 2.5x20 mm stent installation in the distal LAD. The result of TIMI flow 3 angiography evaluation was no dissection or residual stenosis that concluded CAD 1VD post DCA-PCI with 1 Stent in LAD. After the PCI, complaint of chest pain was absent, vital sign examination and physical examinations were within normal limits. The therapy provided consisted of 0.9% NaCl infusion 500cc/24 hours intravenously for hydration after PCI, and dual antiplatelets After day-3 treatment there was no increase in urea and creatinine; therefore, outpatient treatment was carried out followed by phase 2 cardiac rehabilitation.
Keywords: Wellens’ syndrome; typical T wave; stenosis; left anterior descending artery
Abstrak: Sindrom Wellens menggambarkan pola perubahan elektrokardiografi (EKG), terutama gelombang T tipikal yang mengancam keselamatan pasien bila terlambat dilakukan pemeriksaan dan penatalaksanaan lebih lanjut. Kami melaporkan seorang laki-laki beruria 52 tahun dengan riwayat serangan jantung sebelumnya. Pasien memiliki riwayat hipertensi, riwayat hiperkolestrolemia, dan perokok aktif. Tanda vital menunjukkan hemodinamik stabil. Pada pemeriksaan EKG didapatkan gelombang T inversi dalam pada sadapan V2-V6. Pada ekokardiografi didapatkan dimensi ruang jantung normal, LVH konsentrik, dengan disfungsi diastolik grade I, LV EF 52% (SIMPSONS BP), hipokinetik apikal hingga mid anterior, segmen lain normokinetik, MR Mild , dengan kontraktilitas RV normal. Pasien dilakukan intervensi koroner perkutan elektif dengan hasil satu lesi kulprit terdapat pada LAD, stenosis 50-60% proksimal-mid, stenosis tubular 80% distal, stenosis diskret 50-60% D2, dan dilakukan pemasangan stent Promus Premiere 2,5x20mm di distal LAD. Hasil evaluasi angiografi TIMI flow 3, tidak terdapat diseksi, ataupun stenosis residual yang menyimpulkan CAD 1VD post DCA-PCI dengan 1 Stent di LAD. Pasca tindakan, keluhan nyeri dada tidak ada, pemeriksaan tanda vital dan pemeriksaan fisik dalam batas normal. Terapi yang diberikan terdiri dari infus NaCl 0,9% 500cc/24jam intravena untuk hidrasi setelah IKP dan dual antiplatelet. Setelah perawatan hari ke 3 tidak didapatkan peningkatan ureum dan kreatinin, dilakukan rawat jalan yang dilanjutkan dengan rehabilitasi jantung fase 2.
Kata kunci: sindrom Wellens; gelombang T tipikal; stenosis; left anterior descending artery
摘要:韦伦斯综合征描述了一种心电图变化模式,特别是典型的T波,如果延迟进一步检查和管理,则会威胁患者的安全。我们报告了一位52岁男性,既往有心脏病发作史。患者既往有高血压、高胆固醇血症、吸烟史。生命体征显示血流动力学稳定。心电图检查,在V2-V6导联处可见深逆T波。超声心动图,心室尺寸正常,LVH同心,伴有I级舒张功能障碍,左室EF 52% (SIMPSONS BP),心尖至中前段低动力学,其他正常运动节段,MR轻度,右室收缩力正常。患者行选择性经皮冠状动脉介入治疗(PCI),结果发现LAD有1个kulprit病变,近中端狭窄50-60%,远端小管狭窄80%,离散性D2狭窄50-60%,远端LAD安装Promus Premiere 2.5x20 mm支架。TIMI血流3造影评价结果显示无夹层或残留狭窄,结论为LAD DCA-PCI后1支架CAD 1VD。PCI术后无胸痛主诉,生命体征及体格检查均在正常范围内。提供的治疗包括0.9% NaCl静脉输注500cc/24小时用于PCI术后水化,以及双重抗血小板治疗。治疗第3天,尿素和肌酐未升高;因此,先进行门诊治疗,然后进行二期心脏康复。关键词:韦伦斯综合征;典型T波;狭窄;【摘要】慢性肾衰竭患者的左前降支、左前降支、左前降支、左前降支、左前降支、左前降支、左前降支、左前降支、左前降支、左前降支。Kami melaporkan seorang laki-laki beruria 52 tahun dengan riwayat serangan jantung sebelumnya。高血压,高血压性贫血,高血压性贫血,高血压性贫血。坦达vital menunjukkan血液稳定。Pada - preiksaan EKG didapatkan gelombang T反演Pada - sadapan V2-V6。帕达心电图双心室尺寸正常,左心室收缩,登干双心室舒张样I级,左心室EF 52% (simpson BP),心肌动蛋白激酶,中前侧,节段动蛋白正常,MR轻度,登干双心室收缩正常。Pasien dilakukan介入器koroner perkutan elektif dengan hasil satu kulprit terdapat padada LAD,狭窄50-60%前中位,狭窄管80%远端,狭窄diskret 50-60% D2,丹dilakukan pemasangan支架Promus Premiere 2,5 × 20mm远端LAD。血管造影TIMI血流3、血流间隙、血管狭窄残余阳门脉冲、CAD 1VD后DCA-PCI登甘支架1、LAD评价。Pasca tindakan, keluhan nyeri dada tidak ada, pemeriksaan tanda vital, pemeriksaan fisik dalam batas normal。Terapi yang diberikan terdii dari输注NaCl 0.9% 500cc/24jam静脉滴注;Setelah perawatan hari ke 3 tidak didapatkan peningkatan ureum dan kreatiin, dilakukan rawat jalan yang dilanjutkan dengan rehabilitasjantung fase 2。卡塔昆慈:韦伦斯综合症;吉隆邦;狭窄;左前降支