“What Is Actually Being Measured?”: Causality and Underlying Scientific Thinking Process in the Assessment of Depression

IF 2.6 0 PHILOSOPHY
Greta Kaluzeviciute-Moreton
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In clinical psychology, environmental factors such as childhood trauma, chronic stress, social isolation and negative life events (e.g., the loss of a loved one) have been documented as significant risk factors for the development of depression or as trigger events for depressive episodes (Fu & Parahoo, 2009; Kendler, Kuhn, & Prescott, 2004; Neitzke, 2016). Despite this, causal theories of mental health conditions are often tricky and/or scarcely reflected in both diagnostic and rating scales. For instance, the Diagnostic and Statistical Manual of Mental Disorders classifies mental health disorders primarily based on observable symptoms and behaviors, rather than on the underlying psychological and neurobiological processes that may contribute to these symptoms (Kendler, 2006). Similarly, rating scales for depression are typically designed to assess the severity of symptoms, rather than their underlying causes, that is, most rating scales are used as screening tools to identify individuals who may require further evaluation and treatment. Some rating scales may include items related to causal factors, such as the Beck Depression Inventory-II (BDI-II) includes an item that asks about “loss of interest in sex,” which could be related to either biological or psychological factors. Several rating scales include items assessing risk factors for depression, such as family history of depression or personal history of trauma. This may be indicative of both genetic and environmental risk factors, such as the Patient Health [End Page 255] Questionnaire-9, includes an item that asks about family history of depression. However, while risk and causal factors are related, they are distinct in the assessment of depression. Risk factors refer to factors that increase the likelihood of developing depression, while causal factors refer to factors that directly contribute to the development of the condition. The presence of a risk factor does not necessarily mean that an individual will develop depression (Peterson & Seligman, 1984). However, the two are frequently blurred and equated in psychotherapy research (Westen & Bradley, 2005). As such, the historical issues of rating and diagnostic scales, aptly depicted by the Le Moigne (2023) as full of “composite, if not contradictory, heritage, weaving together the classificatory and discontinuous tradition of psychiatry, on the one hand, and the psychometric and continuous tradition of psychology, on the other” (p. XX<EQ>), suffer from an epistemological oversight when it comes to the assessment of causal factors in complex mental health conditions, such as depression. In turn, this contributes to a wider psychometric issue: what is actually being measured? 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引用次数: 0

Abstract

“What Is Actually Being Measured?”: Causality and Underlying Scientific Thinking Process in the Assessment of Depression Greta Kaluzeviciute-Moreton, PhD (bio) Depression is a complex mental health phenomenon due to its multifaceted nature. For one, depression is thought to have a significant genetic component, with studies suggesting that heritability is a significant factor in the development of the disorder (Sullivan, Neale, Kendler, 2000). In clinical psychology, environmental factors such as childhood trauma, chronic stress, social isolation and negative life events (e.g., the loss of a loved one) have been documented as significant risk factors for the development of depression or as trigger events for depressive episodes (Fu & Parahoo, 2009; Kendler, Kuhn, & Prescott, 2004; Neitzke, 2016). Despite this, causal theories of mental health conditions are often tricky and/or scarcely reflected in both diagnostic and rating scales. For instance, the Diagnostic and Statistical Manual of Mental Disorders classifies mental health disorders primarily based on observable symptoms and behaviors, rather than on the underlying psychological and neurobiological processes that may contribute to these symptoms (Kendler, 2006). Similarly, rating scales for depression are typically designed to assess the severity of symptoms, rather than their underlying causes, that is, most rating scales are used as screening tools to identify individuals who may require further evaluation and treatment. Some rating scales may include items related to causal factors, such as the Beck Depression Inventory-II (BDI-II) includes an item that asks about “loss of interest in sex,” which could be related to either biological or psychological factors. Several rating scales include items assessing risk factors for depression, such as family history of depression or personal history of trauma. This may be indicative of both genetic and environmental risk factors, such as the Patient Health [End Page 255] Questionnaire-9, includes an item that asks about family history of depression. However, while risk and causal factors are related, they are distinct in the assessment of depression. Risk factors refer to factors that increase the likelihood of developing depression, while causal factors refer to factors that directly contribute to the development of the condition. The presence of a risk factor does not necessarily mean that an individual will develop depression (Peterson & Seligman, 1984). However, the two are frequently blurred and equated in psychotherapy research (Westen & Bradley, 2005). As such, the historical issues of rating and diagnostic scales, aptly depicted by the Le Moigne (2023) as full of “composite, if not contradictory, heritage, weaving together the classificatory and discontinuous tradition of psychiatry, on the one hand, and the psychometric and continuous tradition of psychology, on the other” (p. XX), suffer from an epistemological oversight when it comes to the assessment of causal factors in complex mental health conditions, such as depression. In turn, this contributes to a wider psychometric issue: what is actually being measured? Le Moigne’s detailed overview predominantly focuses on “superficial” symptomatology of depression (which contributed to two distinct forms of evaluation: hetero- and self-evaluation), and its subsequent impact on the classification and taxonomy of depression throughout history. However, the overarching conceptualization of depression (including a more detailed consideration of causal factors and their assessment) and the usefulness of rating scales in applied research (such as psychotherapy) remain in the background. The question of “what is actually being measured” is obviously not new in psychometrics. However, it is important to continuously revisit this issue, given that rating scales (such as the BDI) are considered to be universally applicable “evidence-based” tools (Rogers, Adler, Bungay, & Wilson, 2005) as well as a common and well-accepted form of scientific thinking in psychotherapy research (Kaluzeviciute & Willemsen, 2020). From an epistemological point of view, a scientific thinking style entails not only cognitive operations common to methodology, but also practices of generating, sharing, assimilating and transforming knowledge (Hacking, 2012). Therefore, it is important to acknowledge that any scientific tool, including rating scales, encompasses not just what but also how researchers find out about their studied phenomena (i.e., a scientific thinking style conveys a performative quality) (Kaluzeviciute & Willemsen, 2020). As identified by Le Moigne (2023), taxonomy leads toward a category-based logic in that it considers the examined phenomenon to be shared by only a...
“实际被衡量的是什么?”:抑郁症评估中的因果关系和潜在的科学思维过程
“实际被衡量的是什么?”:抑郁症的因果关系和潜在的科学思维过程Greta Kaluzeviciute-Moreton博士(生物)抑郁症是一种复杂的心理健康现象,由于其多方面的性质。首先,抑郁症被认为具有重要的遗传成分,研究表明,遗传性是该疾病发展的重要因素(Sullivan, Neale, Kendler, 2000)。在临床心理学中,环境因素,如童年创伤、慢性压力、社会孤立和负面生活事件(如失去亲人)已被记录为抑郁症发展的重要风险因素或抑郁症发作的触发事件(Fu & Parahoo, 2009;肯德勒,库恩和普雷斯科特,2004;Neitzke, 2016)。尽管如此,心理健康状况的因果理论往往是棘手的和/或几乎没有反映在诊断和评级量表。例如,《精神障碍诊断和统计手册》主要根据可观察到的症状和行为对精神健康障碍进行分类,而不是根据可能导致这些症状的潜在心理和神经生物学过程(Kendler, 2006年)。同样,抑郁症的评定量表通常用于评估症状的严重程度,而不是其潜在原因,也就是说,大多数评定量表被用作筛选工具,以确定可能需要进一步评估和治疗的个体。一些评定量表可能包括与因果因素相关的项目,如贝克抑郁量表ii (BDI-II)包括一个关于“对性失去兴趣”的项目,这可能与生物或心理因素有关。一些评定量表包括评估抑郁症风险因素的项目,如抑郁症家族史或个人创伤史。这可能是遗传和环境风险因素的指示,如患者健康问卷-9,包括一个关于抑郁症家族史的问题。然而,虽然风险因素和因果因素是相关的,但它们在抑郁症的评估中是不同的。风险因素是指增加患抑郁症可能性的因素,而因果因素是指直接导致病情发展的因素。风险因素的存在并不一定意味着个体会患上抑郁症(Peterson & Seligman, 1984)。然而,在心理治疗研究中,这两者经常被模糊和等同起来(Westen & Bradley, 2005)。因此,评定和诊断量表的历史问题,被Le Moigne(2023)恰当地描述为“一方面是精神病学的分类和不连续的传统,另一方面是心理学的心理测量学和连续的传统,交织在一起,即使不是矛盾的,遗产是复合的”(第XX页),在评估复杂心理健康状况(如抑郁症)的因果因素时,受到认识论的忽视。反过来,这又引出了一个更广泛的心理测量问题:到底测量的是什么?Le Moigne的详细概述主要集中在抑郁症的“表面”症状学(这促成了两种截然不同的评估形式:异质评估和自我评估),以及它在整个历史上对抑郁症分类和分类的后续影响。然而,抑郁症的总体概念(包括对因果因素及其评估的更详细的考虑)和评定量表在应用研究(如心理治疗)中的有用性仍然是背景。“实际测量的是什么”这个问题显然在心理测量学中并不新鲜。然而,考虑到评定量表(如BDI)被认为是普遍适用的“循证”工具(Rogers, Adler, Bungay, & Wilson, 2005),以及心理治疗研究中常见且被广泛接受的科学思维形式(Kaluzeviciute & Willemsen, 2020),不断重新审视这个问题是很重要的。从认识论的角度来看,科学思维方式不仅需要方法论共同的认知操作,还需要生成、共享、吸收和转化知识的实践(Hacking, 2012)。因此,重要的是要承认,任何科学工具,包括评分量表,不仅包括研究人员发现他们所研究的现象的内容,还包括研究人员如何发现他们所研究的现象(即,科学的思维方式传达了一种行为品质)(Kaluzeviciute & Willemsen, 2020)。正如Le Moigne(2023)所指出的那样,分类学导致了一种基于类别的逻辑,因为它认为所检查的现象仅由一个…
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CiteScore
3.60
自引率
4.30%
发文量
40
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