Sleep Disruption in a Mouse Model of Chronic Traumatic Brain Injury

Andrew R Morris, Erwin K Gudenschwager Basso, Miguel A Gutierrez-Monreal, Rawad Daniel Arja, Firas H Kobeissy, Christopher G Janus, Kevin KW Wang, Jiepei Zhu, Andrew C Liu
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Abstract

Chronic sleep/wake disturbances are strongly associated with traumatic brain injury (TBI) in patients and are being increasingly recognized. However, the underlying mechanisms are largely understudied and there is an urgent need for animal models of lifelong sleep/wake disturbances. The objective of this study was to develop a chronic TBI rodent model and investigate the lifelong chronic effect of TBI on sleep/wake behavior. We performed repetitive midline fluid percussion injury (rmFPI) in four months old mice and monitored their sleep/wake behavior using the non-invasive PiezoSleep system. The sleep/wake states were recorded before injury (baseline) and then monthly thereafter. We found that TBI mice displayed a significant decrease in sleep duration in both the light and dark phases, beginning at three months post-TBI and continuing throughout the study. Consistent with the sleep phenotype, these TBI mice showed circadian locomotor activity phenotypes and exhibited reduced anxiety-like behavior. TBI mice also gained less weight, and had less lean mass and total body water content, compared to sham controls. Furthermore, TBI mice showed extensive brain tissue loss and increased GFAP and IBA1 levels in the hypothalamus and the vicinity of the injury, indicative of chronic neuropathology. In summary, our study identified a critical time window of TBI pathology and associated circadian and sleep/wake phenotypes. Future studies should leverage this mouse model to investigate the molecular mechanisms underlying the chronic sleep/wake phenotypes following TBI early in life.
慢性创伤性脑损伤小鼠模型的睡眠中断
慢性睡眠/觉醒障碍与患者的创伤性脑损伤(TBI)密切相关,并且越来越被认识到。然而,潜在的机制在很大程度上还没有得到充分的研究,迫切需要建立终身睡眠/觉醒障碍的动物模型。本研究的目的是建立慢性创伤性脑损伤啮齿动物模型,研究创伤性脑损伤对睡眠/觉醒行为的终身慢性影响。我们对四个月大的小鼠进行重复性中线液体冲击损伤(rmFPI),并使用无创PiezoSleep系统监测它们的睡眠/觉醒行为。在受伤前记录睡眠/清醒状态(基线),之后每月记录一次。我们发现,创伤性脑损伤小鼠在光明期和黑暗期的睡眠时间都显着减少,从创伤性脑损伤后三个月开始,并在整个研究过程中持续下去。与睡眠表型一致,这些TBI小鼠表现出昼夜运动活动表型,并表现出减少的焦虑样行为。与假对照组相比,TBI小鼠体重增加更少,瘦质量和全身含水量也更少。此外,TBI小鼠表现出广泛的脑组织丢失,下丘脑和损伤附近的GFAP和IBA1水平升高,表明慢性神经病理。总之,我们的研究确定了TBI病理和相关的昼夜节律和睡眠/觉醒表型的关键时间窗。未来的研究应该利用这种小鼠模型来研究早期TBI后慢性睡眠/觉醒表型的分子机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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