Production of interleukin 2 (IL-2) and responsiveness to IL-2 of peripheral blood lymphocytes in minimal change nephrotic syndrome.

K Dohi, H Morita, S Ogawa, T Hirayama, H Yamada, H Ishikawa
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引用次数: 5

Abstract

We investigated the role cell-mediated immunity in minimal change nephrotic syndrome (MCNS) by measuring interleukin 2 (IL-2) production and the responsiveness to IL-2 of peripheral blood lymphocytes (PBL). PBL from patients with MCNS, who were in the nephrotic stage prior to initiation of prednisolone (PSL) treatment or who were in remission for less than 1 yr, exhibited significantly lower levels of IL-2 production. In contrast, PBL from patients with MCNS, who were in remission for more than 1 yr or who could remit from the PSL regimen, showed normal IL-2 production. IL-2 production by CD4+ cells from patients with MCNS in the nephrotic stage was normal, but that by CD8+ cells was markedly reduced, however returned to normal when the disease was in remission. The responsiveness to exogenous IL-2 of concanavalin A-induced lymphoblasts from patients with MCNS was significantly lower, although the proportion of Tac antigen-positive cells did not differ from that of healthy volunteers. These findings suggest that defective IL-2 production and IL-2 responsiveness of PBL in patients with MCNS contribute to the pathogenesis of MCNS.

微小变化肾病综合征外周血淋巴细胞白细胞介素2 (IL-2)的产生和对IL-2的反应。
我们通过测量白细胞介素2 (IL-2)的产生和外周血淋巴细胞(PBL)对IL-2的反应性来研究细胞介导免疫在微小改变肾病综合征(MCNS)中的作用。MCNS患者在开始强的松龙(PSL)治疗前处于肾病期或缓解期少于1年的PBL, IL-2产生水平显著降低。相比之下,缓解期超过1年或可以从PSL方案中缓解的MCNS患者的PBL显示正常的IL-2生成。MCNS患者肾病期CD4+细胞产生的IL-2是正常的,但CD8+细胞产生的IL-2明显减少,但在疾病缓解时恢复正常。MCNS患者的豆豆蛋白a诱导淋巴细胞对外源性IL-2的反应性明显降低,尽管Tac抗原阳性细胞的比例与健康志愿者没有差异。这些发现提示MCNS患者白细胞介素-2产生缺陷和白细胞介素-2反应性缺陷参与了MCNS的发病机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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