A Presentation of Persistent Pulmonary Hypertension of a Newborn Mimicking Coarctation of Aorta

Chang-Hsien Yu
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Abstract

A male newborn was born to a 18y/o mother at gestational age of 38weeks, birth weight 3295gm via emergent Cesarean section with the indication of fetal distress. The mother was diagnosed with type II DM and pregnancy-induced hypertension. Aspirin was given by a family doctor for prevention of preeclampsia during gestational age of 29 to 36 weeks. After birth, the baby presented persistent desaturation (SpO2 77-85%) and severe respiratory acidosis despite of resuscitation. CXR revealed cardiomegaly. Echocardiography showed remarkable dilatation of right ventricle and a huge Patent Ductus of Arteriosus (PDA) with almost right-toleft shunt. Persistent Pulmonary Hypertension of Newborn (PPHN) was diagnosed. However, the isthmus of aorta was restricted (the narrowest diameter was 0.220cm) (Figure 1).
模拟主动脉缩窄的新生儿持续性肺动脉高压的表现
一名18岁母亲在妊娠期38周,出生体重3295克,经紧急剖宫产分娩,新生儿为男婴。母亲被诊断为II型糖尿病和妊娠高血压。阿斯匹林是由家庭医生在妊娠29至36周期间用于预防先兆子痫。出生后,尽管复苏,婴儿仍出现持续的去饱和(SpO2 77-85%)和严重的呼吸性酸中毒。CXR显示心脏肿大。超声心动图显示右心室明显扩张,动脉导管未闭(PDA)巨大,几乎为右至左分流。诊断为新生儿持续性肺动脉高压(PPHN)。然而,主动脉峡部受限(最窄直径0.220cm)(图1)。
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