Patterns of activation in ventricular arrhythmias of late myocardial infarction in dogs.

European journal of cardiology Pub Date : 1979-11-01
E Kaplinsky, S Ogawa, J Kmetzo, L S Dreifus
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引用次数: 0

Abstract

Ventricular arrhythmias were produced in 12 dogs 4 to 6 days after coronary artery ligation by programmed ventricular stimulation. The electrocardiogram and 7 composite electrograms from endocardial and epicardial surfaces of the ischemic, border and normal zones, as well as from the right ventricle, were recorded during and after programmed ventricular stimulation. The ventricular arrhythmias were preceded and sustained by delayed, fragmented activity in the ischemic epicardial zone bridging diastole. Efferent pathways from the ischemic epicardium led to direct epicardial spread to adjacent normal epicardium in most instances. Efferent pathways into the endocardial regions were also observed, but to a lesser extent. The efferent reentry pathways led to both ventricles, and produced right and left ventricular arrhythmias in 8 of the 12 dogs; they were exclusively of left ventricular origin in the remaining 5. Classification of right and left ventricular arrhythmias may only be related to the exit points and not necessarily to their origin.

犬心肌梗死晚期室性心律失常的激活模式。
12只狗在冠状动脉结扎术后4 ~ 6天发生室性心律失常。记录程序性心室刺激前后心内膜、心外膜缺血区、边缘区和正常区以及右心室的心电图和7个复合心电图。室性心律失常是由缺血心外膜带桥接舒张的延迟、碎片化活动引起的。在大多数情况下,缺血心外膜的传出通路导致心外膜直接扩散到邻近的正常心外膜。心内膜区域的传出通路也被观察到,但程度较轻。传出再入通路通向两个心室,并在12只狗中产生8只左右心室心律失常;其余5例均为左心室起源。左右室性心律失常的分类可能只与出口点有关,而不一定与起源点有关。
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