Myeloid-derived TGFβ1 and oncostatin M establish two distinct stromal niches controlling late and hybrid EMT states.

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DOI:10.1038/s43018-026-01259-3
作者：Anqi Dong,?Sebastiaan Vanuytven,?Andrea Pérez González,?Kevin Bévant,?Lieselot Van de Putte,?Zahra Zahedi,?Justine Allard,?Christine Dubois,?Isabelle Salmon,?Sandrine Rorive,?Cédric Blanpain
文献类型：期刊论文
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Nature

Abstract：Epithelial-to-mesenchymal transition (EMT) regulates tumor progression, metastasis and resistance to therapy. EMT encompasses a continuum of tumor states displaying different functions. The

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