Amorphous silica nanoparticles caused lung injury through the induction of epithelial apoptosis via ROS/Ca²⁺/DRP1-mediated mitochondrial fission signaling.

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DOI:10.1080/17435390.2022.2144774
作者：Yan Li,?Yawen Zhu,?Bosen Zhao,?Qing Yao,?Hailin Xu,?Songqing Lv,?Ji Wang,?Zhiwei Sun,?Yanbo Li,?Caixia Guo
文献类型：期刊论文
补充材料：只需要正文
文献链接：https://doi.org/10.1080/17435390.2022.2144774

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Abstract：The adverse effects of amorphous silica nanoparticles (SiNPs) exposure on the respiratory system were increasingly recognized, however, its potential pathogenesis still remains not fully elu

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Amorphous silica nanoparticles caused lung injury through the induction of epithelial apoptosis via ROS/Ca2+/DRP1-mediated mitochondrial fission signaling.

Amorphous silica nanoparticles caused lung injury through the induction of epithelial apoptosis via ROS/Ca²⁺/DRP1-mediated mitochondrial fission signaling.