A mitochondrial surveillance mechanism activated by SRSF2 mutations in hematologic malignancies.

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DOI:10.1172/jci175619
文献类型：期刊论文
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Journal of Clinical Investigation (JCI)

Abstract：Splicing factor mutations are common in myelodysplastic syndrome (MDS) and acute myeloid leukemia (AML), but how they alter cellular functions is unclear. We show that the pathogenic SRSF2P9

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