Intestinal-specific ketohexokinase deficiency ameliorates high fructose-induced metabolic dysfunction across the intestinal-liver-kidney axis in mice

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DOI:10.1016/j.jnutbio.2026.110453
作者：Shanshan Song,?Xiaoli Yi,?Mulan Wang,?Huiru Yang,?Wenting Tan,?Ying Zhu,?Jun Yu,?Chuanming Xu
文献类型：期刊论文
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Elsevier

Abstract：The small intestine, one of the three major sites for ketohexokinase (KHK, also known as fructokinase) expression, plays a crucial role in fructose-related pathological processes, primarily

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