P. Kraiczy, K. Hunfeld, R. Würzner, G. Acker, V. Brade
{"title":"早期莱姆病血清对补体耐药阿兹利螺旋体FEM1野生型和缺乏ospc的FEM1变体的杀螺旋体活性研究","authors":"P. Kraiczy, K. Hunfeld, R. Würzner, G. Acker, V. Brade","doi":"10.1016/S0162-3109(00)80225-8","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":13327,"journal":{"name":"Immunopharmacology","volume":"6 1","pages":"75"},"PeriodicalIF":0.0000,"publicationDate":"2000-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Studies on the borreliacidal activity of early lyme disease sera against complement-resistant borrelia afzelii FEM1 wild-type and an OspC-lacking FEM1 variant\",\"authors\":\"P. Kraiczy, K. Hunfeld, R. Würzner, G. Acker, V. Brade\",\"doi\":\"10.1016/S0162-3109(00)80225-8\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\",\"PeriodicalId\":13327,\"journal\":{\"name\":\"Immunopharmacology\",\"volume\":\"6 1\",\"pages\":\"75\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2000-08-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Immunopharmacology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1016/S0162-3109(00)80225-8\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Immunopharmacology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1016/S0162-3109(00)80225-8","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Studies on the borreliacidal activity of early lyme disease sera against complement-resistant borrelia afzelii FEM1 wild-type and an OspC-lacking FEM1 variant