W. Liszewski, David Gram Naym, E. Biskup, R. Gniadecki
{"title":"I型干扰素通过JAK1-STAT1途径增强补骨脂素与紫外线A诱导的皮肤淋巴瘤细胞系凋亡的关系","authors":"W. Liszewski, David Gram Naym, E. Biskup, R. Gniadecki","doi":"10.1111/phpp.12302","DOIUrl":null,"url":null,"abstract":"Photochemotherapy with psoralen and ultraviolet A (PUVA), with or without adjuvant interferon‐α (IFN‐α), is a first‐line therapy for early‐stage mycosis fungoides and other forms of cutaneous T‐cell lymphoma (CTCL). However, the mechanism by which PUVA with IFN‐α work in CTCL is poorly understood.","PeriodicalId":20060,"journal":{"name":"Photodermatology","volume":"36 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2017-04-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"13","resultStr":"{\"title\":\"Psoralen with ultraviolet A‐induced apoptosis of cutaneous lymphoma cell lines is augmented by type I interferons via the JAK1–STAT1 pathway\",\"authors\":\"W. Liszewski, David Gram Naym, E. Biskup, R. Gniadecki\",\"doi\":\"10.1111/phpp.12302\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Photochemotherapy with psoralen and ultraviolet A (PUVA), with or without adjuvant interferon‐α (IFN‐α), is a first‐line therapy for early‐stage mycosis fungoides and other forms of cutaneous T‐cell lymphoma (CTCL). However, the mechanism by which PUVA with IFN‐α work in CTCL is poorly understood.\",\"PeriodicalId\":20060,\"journal\":{\"name\":\"Photodermatology\",\"volume\":\"36 1\",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2017-04-17\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"13\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Photodermatology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1111/phpp.12302\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Photodermatology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1111/phpp.12302","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Psoralen with ultraviolet A‐induced apoptosis of cutaneous lymphoma cell lines is augmented by type I interferons via the JAK1–STAT1 pathway
Photochemotherapy with psoralen and ultraviolet A (PUVA), with or without adjuvant interferon‐α (IFN‐α), is a first‐line therapy for early‐stage mycosis fungoides and other forms of cutaneous T‐cell lymphoma (CTCL). However, the mechanism by which PUVA with IFN‐α work in CTCL is poorly understood.
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