致癌过程中再分布趋化因子作用的不一致生物学

L. Agius
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引用次数: 0

摘要

增殖的肿瘤细胞所表现出的促进性抗原性谱允许一个允许的微环境,正如未成熟DC所预测的那样。在重新定义的趋化因子反应性环境中,抗原刺激的重新分配允许整体免疫系统的预测调节,正如重新定义的趋化因子反应性所提出的那样。正是多种抗原性的简单并置,使得免疫耐受性和容许性得以出现。这种抗原性的重新表征是对固有成分途径反应性的重新表征,实际上是在对增殖肿瘤细胞床的免疫反应谱中预测的趋化因子重新分配。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Incongruent Biology of Redistributed Chemokine Action in Carcinogenesis
The promotional antigenicity profiles exhibited by proliferating tumor cells allows for a permissive microenvironment as projected by immature DC. It is within redefined milieu of chemokine reactivity that redistribution of antigenic stimuli allow for projected modulations of the integral immune systems as proposed by the redefined ch emokine reactivity. It is the simple juxtaposition of multiple range of antigenicity that perforce allows for immune tolerance and permissiveness to emerge. The recharacterization of such antigenicity is recharacterization of inherent component pathway reactivity as indeed projected chemokine redistribution within profiles of the immune response to proliferating tumor cell beds.
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