{"title":"PGF2α对血管组织钙通量和收缩的作用及其阻断剂ca++进入阻断剂(Ca拮抗剂)","authors":"T. Godraind, R. Miller","doi":"10.1016/0090-6980(84)90278-8","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":83897,"journal":{"name":"Research in prostaglandins","volume":"287 1","pages":"33"},"PeriodicalIF":0.0000,"publicationDate":"1984-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"1","resultStr":"{\"title\":\"Abstract of the NATO Advanced Research Workshop on Icosanoids and Ion transport November 17–18, 1983 ParisThe action of PGF2α on Ca fluxes and contraction in vascular tissue and their blockade CA++ entry blockers (Ca antagonists)\",\"authors\":\"T. Godraind, R. Miller\",\"doi\":\"10.1016/0090-6980(84)90278-8\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\",\"PeriodicalId\":83897,\"journal\":{\"name\":\"Research in prostaglandins\",\"volume\":\"287 1\",\"pages\":\"33\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1984-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"1\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Research in prostaglandins\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1016/0090-6980(84)90278-8\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Research in prostaglandins","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1016/0090-6980(84)90278-8","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Abstract of the NATO Advanced Research Workshop on Icosanoids and Ion transport November 17–18, 1983 ParisThe action of PGF2α on Ca fluxes and contraction in vascular tissue and their blockade CA++ entry blockers (Ca antagonists)