【收回】CCL2 siRNA对U251人胶质瘤细胞系增殖和凋亡的影响。

IF 3.4 3区 医学 Q2 MEDICINE, RESEARCH & EXPERIMENTAL
Molecular medicine reports Pub Date : 2024-01-01 Epub Date: 2023-11-08 DOI:10.3892/mmr.2023.13124
Bin Lu, Yue Zhou, Zhongzhou Su, Ai Yan, Peng Ding
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引用次数: 0

摘要

这篇论文发表后,一位关心此事的读者提请编辑注意,第3392页图4A所示的VEGF蛋白质印迹数据与不同研究机构的不同作者撰写的其他文章中以不同形式出现的数据惊人地相似。由于上述文章中有争议的数据在提交给《分子医学报告》之前已经在其他地方发表,编辑决定将这篇论文从《分子医学杂志》上撤回。作者被要求对这些担忧做出解释,但编辑部从未收到回复。对于由此给读者带来的不便,编辑深表歉意。【分子医学报告16:3387-33942017;DOI:10.3892/mmr.2017.6995】。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Retracted] Effect of CCL2 siRNA on proliferation and apoptosis in the U251 human glioma cell line.

Following the publication of this paper, it was drawn to the Editor's attention by a concerned reader that the VEGF western blotting data shown in Fig. 4A on p. 3392 were strikingly similar to data appearing in different form in other articles written by different authors at different research institutes.  Owing to the fact that the contentious data in the above article had already been published elsewhere prior to its submission to Molecular Medicine Reports, the Editor has decided that this paper should be retracted from the Journal. The authors were asked for an explanation to account for these concerns, but the Editorial Office never received a reply.The Editor apologizes to the readership for any inconvenience caused. [Molecular Medicine Reports 16: 3387‑3394, 2017; DOI: 10.3892/mmr.2017.6995].

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来源期刊
Molecular medicine reports
Molecular medicine reports 医学-病理学
CiteScore
7.60
自引率
0.00%
发文量
321
审稿时长
1.5 months
期刊介绍: Molecular Medicine Reports is a monthly, peer-reviewed journal available in print and online, that includes studies devoted to molecular medicine, underscoring aspects including pharmacology, pathology, genetics, neurosciences, infectious diseases, molecular cardiology and molecular surgery. In vitro and in vivo studies of experimental model systems pertaining to the mechanisms of a variety of diseases offer researchers the necessary tools and knowledge with which to aid the diagnosis and treatment of human diseases.
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