实验性辐射暴露后肺不张的发展

Q4 Medicine

Clinical and Experimental Morphology Pub Date : 2020-01-01 DOI:10.31088/cem2020.9.1.30-39

Y. Kirillov, I. Chernov, E. Malysheva, S. Timofeev, E. Startseva, O. Rozenberg

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引用次数: 1

摘要

结果。实验早期发生的肺不张与表面活性剂有关，这可以通过灌洗液中PS和PL的减少得到证实。晚期肺不张的形成是由于上皮内膜增厚导致支气管和细支气管的管腔闭塞、狭窄和明显的支气管收缩引起的。结论。了解肺不张发展的机制对于预防上述病理和减少并发症的风险是必要的。

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Development of pulmonary atelectasis after experimental radiation exposure

Results . Atelectasis occurred at the early stages of the experiment is surfactant-dependent, which is confirmed by the decreased amount of PS and PL in lavage fluid. The formation of atelectasis in later periods is caused by the obturation, narrowing of the lumen of the bronchi and bronchioles due to thickening of the epithelial lining and pronounced bronchial bronchoconstriction. Conclusion. Understanding the mechanisms of development of atelectasis is necessary in order to prevent the pathology described above and reduce the risk of complications.

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来源期刊

Clinical and Experimental Morphology Biochemistry, Genetics and Molecular Biology-Cancer Research

CiteScore

0.60

自引率

0.00%

发文量