氨和亚硝酸盐对不同年龄太平洋大头鳕鱼(Gadus macrocephalus)幼虫的急性毒性

Q3 Chemical Engineering
Wei Wang, Hua Wang, Chun-Man Yu, Zhiqiang Jiang
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引用次数: 15

摘要

摘要本试验研究了氨和亚硝酸盐对太平洋大头鳕(Gadus macrocephalus) 3个发育阶段(孵化后11、22和35 d,平均总长度分别为4.63±0.14、5.83±0.17和7.46±0.23 mm)幼虫的急性毒性。结果首次表明,氨和亚硝酸盐的急性毒性与太平洋鳕鱼幼虫的年龄密切相关,氨和亚硝酸盐的急性毒性随太平洋鳕鱼幼虫生长的增加而增加。孵化后11天、22天和35天的太平洋鳕鱼仔鱼暴露48 h后,未电离氨氮(UIAN)的致死浓度(LC50)分别为1.72、0.69和0.32 mg L−1。孵化后11 d、22 d和35 d亚硝酸盐氮对太平洋鳕鱼仔鱼的48 h LC50分别为831.76、269.15和223.87 mg L−1。结果表明,氨对太平洋鳕鱼幼鱼的急性毒性远高于亚硝酸盐。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Acute toxicity of ammonia and nitrite to different ages of Pacific cod (Gadus macrocephalus) larvae
Abstract This study was conducted to evaluate the acute toxicity of ammonia and nitrite to three developmental stages of Pacific cod (Gadus macrocephalus) larvae (11, 22, and 35 days after hatching, with mean total lengths of 4.63 ± 0.14, 5.83 ± 0.17, and 7.46 ± 0.23 mm, respectively). The results showed for the first time that the acute toxicity of ammonia and nitrite is closely related to the age of Pacific cod larvae, and the acute toxicity of ammonia or nitrite increased with increased Pacific cod larval growth. Lethal concentrations (LC50) of un-ionized ammonia nitrogen (UIAN) for a 48-h exposure in 11-day post-hatch, 22-day post-hatch, and 35-day post-hatch Pacific cod larvae were 1.72, 0.69, and 0.32 mg L−1, respectively. The 48-h LC50 of nitrite nitrogen to Pacific cod larvae 11-day post-hatch, 22-day post-hatch, and 35-day post-hatch were 831.76, 269.15, and 223.87 mg L−1, respectively. The present findings demonstrate that the acute toxicity of ammonia for Pacific cod larvae is much higher than that of nitrite.
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来源期刊
CiteScore
1.62
自引率
0.00%
发文量
0
审稿时长
1 months
期刊介绍: Chemical Speciation & Bioavailability ( CS&B) is a scholarly, peer-reviewed forum for insights on the chemical aspects of occurrence, distribution, transport, transformation, transfer, fate, and effects of substances in the environment and biota, and their impacts on the uptake of the substances by living organisms. Substances of interests include both beneficial and toxic ones, especially nutrients, heavy metals, persistent organic pollutants, and emerging contaminants, such as engineered nanomaterials, as well as pharmaceuticals and personal-care products as pollutants. It is the aim of this Journal to develop an international community of experienced colleagues to promote the research, discussion, review, and spread of information on chemical speciation and bioavailability, which is a topic of interest to researchers in many disciplines, including environmental, chemical, biological, food, medical, toxicology, and health sciences. Key themes in the scope of the Journal include, but are not limited to, the following “6Ms”: Methods for speciation analysis and the evaluation of bioavailability, especially the development, validation, and application of novel methods and techniques. Media that sustain the processes of release, distribution, transformation, and transfer of chemical speciation; of particular interest are emerging contaminants, such as engineered nanomaterials, pharmaceuticals, and personal-care products. Mobility of substance species in environment and biota, either spatially or temporally. Matters that influence the chemical speciation and bioavailability, mainly environmentally relevant conditions. Mechanisms that govern the transport, transformation, transfer, and fate of chemical speciation in the environment, and the biouptake of substances. Models for the simulation of chemical speciation and bioavailability, and for the prediction of toxicity. Chemical Speciation & Bioavailability is a fully open access journal. This means all submitted articles will, if accepted, be available for anyone to read, anywhere, at any time. immediately on publication. There are no charges for submission to this journal.
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