P16的过表达通过激活ERK1/2信号通路逆转mdr1介导的宫颈腺癌DDP耐药

IF 2.8 4区生物学 Q3 CELL BIOLOGY

Cell Division Pub Date : 2019-07-06 DOI:10.1186/s13008-019-0048-6

Yun Xiao, M. Liang, Cheng-cheng Liu, Yanan Wang, Yang Zeng, Jun Zhou, Hui-ting Zhu, Qin Wang, Y. Zou, S. Zeng

引用次数: 5

摘要

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Overexpression of P16 reversed the MDR1-mediated DDP resistance in the cervical adenocarcinoma by activating the ERK1/2 signaling pathway

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来源期刊

Cell Division CELL BIOLOGY-

CiteScore

3.70

自引率

0.00%

发文量

审稿时长

>12 weeks

期刊介绍： Cell Division is an open access, peer-reviewed journal that encompasses all the molecular aspects of cell cycle control and cancer, cell growth, proliferation, survival, differentiation, signalling, gene transcription, protein synthesis, genome integrity, chromosome stability, centrosome duplication, DNA damage and DNA repair. Cell Division provides an online forum for the cell-cycle community that aims to publish articles on all exciting aspects of cell-cycle research and to bridge the gap between models of cell cycle regulation, development, and cancer biology. This forum is driven by specialized and timely research articles, reviews and commentaries focused on this fast moving field, providing an invaluable tool for cell-cycle biologists. Cell Division publishes articles in areas which includes, but not limited to: DNA replication, cell fate decisions, cell cycle & development Cell proliferation, mitosis, spindle assembly checkpoint, ubiquitin mediated degradation DNA damage & repair Apoptosis & cell death