Erb‐(IL10)2通过消除氧自由基改善辐射诱导的皮肤损伤

Q4 Medicine
Jiahe Xu, Jiaxing Zhu, Qi Zhao, J. Xue, S. Qin
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引用次数: 0

摘要

放射性皮肤损伤(RISI)在放射治疗中仍然是一个严重的问题。IL-10被认为是一种免疫抑制性细胞因子,通过抑制细胞中促炎细胞因子的分泌。本研究的目的是评估Erb(IL10)2对电离辐射的保护作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Erb‐(IL10)2 ameliorates radiation‐induced skin injury through eliminate oxygen free radicals
Radiation‐induced skin injury (RISI) remains a serious concern during radiotherapy. IL‐10 is considered as an immune suppressive cytokine by inhibiting the secretion of the proinflammatory cytokines in cells. The aim of this study was to evaluate the protective role of Erb (IL10) 2 against ionizing radiation.
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来源期刊
Precision Radiation Oncology
Precision Radiation Oncology Medicine-Oncology
CiteScore
1.20
自引率
0.00%
发文量
32
审稿时长
13 weeks
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