通过诱导酶抑制剂控制肥胖的高脂肪食品综述

D. R. Sari, Aurelia Afra, Erni Yupita Sari Br Sembiring, Cico Jhon Karunia Simamora
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引用次数: 0

摘要

背景:肥胖是由于身体脂肪组织过多而导致的身高和体重失衡。撰写这篇综述的目的是找出酶抑制剂诱导对富含脂肪的食物作为控制肥胖的作用。方法:采用文献研究方法,对与源问题相关的问题进行写作和评估。结果:控制肥胖的一种方法是通过调节饮食模式和脂肪酶抑制剂的消耗。抑制脂肪酶是饮食医学中发展最广泛的有效方法之一。抑制性化合物导致胰脂肪酶失去分解能力,进入血液。植物来源的脂肪酶抑制剂化合物的效力可以在数量和性能上都得到提高。通过微生物的发酵来增加次级代谢产物组抑制剂的产生。结论:通过抑制脂肪酶抑制甘油三酯水解,可以减少和预防肥胖。次生代谢产物的诱导可以用微生物发酵。药用植物中次生代谢产物化合物的产生可以在发酵的情况下增加。黄酮类化合物可以减少脂质在心脏中的积累,减少葡萄糖的吸收,抑制多糖分解为单糖。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Fat-Rich Food Review on Obesity Control through Induction Enzyme Inhibitors
Background: Obesity is an imbalance between height and weight due to excessive body fat tissue. The purpose of writing this review is to find out the effect of enzyme inhibitor induction on fat-rich foods as control of obesity. Method: Writing and assessing source problems related to using literature study methods. Results: One way of controlling obesity is by regulating dietary patterns and consumption of lipase inhibitors. Inhibition of lipase is one of the most widely developed effective ways in diet medicine. Inhibitory compounds cause pancreatic lipase to lose its ability in decomposition that enters the blood. The potency of plant-origin lipase inhibitor compounds can be increased in both number and performance. Increasing the production of secondary metabolite group inhibitors is by fermentation of microorganisms. Conclusion: Inhibition of triglyceride hydrolysis through inhibition of lipase enzymes can decrease and prevent obesity. Secondary metabolite induction can be fermented with microorganisms. The production of secondary metabolite compounds in medicinal plants can be increased in the presence of fermentation. Flavonoids can decrease the accumulation of lipids in the heart, reduce glucose absorption, inhibit the breakdown of polysaccharides into monosaccharides.
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