谷氨酸依赖假说可以解释过度食用棕色食物和世界范围内的心脏病流行

J. Cocores
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引用次数: 1

摘要

接下来的研究试图确定含有美拉德反应最终产物的食物是否比不含美拉德反应最终产物的食物更有吸引力、更有回报、更容易上瘾、更容易引起心血管疾病。同样重要的是,与不含美拉德反应最终产物的食品相比,含有美拉德反应最终产物的食品是否表现出更强的耐受性(通过每天增加的总热量消耗来衡量),通过增加超重量来衡量的进展,以及通过更频繁地渴望吃零食和吃饭来衡量的戒断?本文提供的数据表明,饮食美拉德反应终产物诱导的谷氨酸依赖、过度食用棕色食物和心血管疾病之间存在密切联系。磷和谷氨酸引起的氧化还原失衡、高脂血症、免疫系统激活、慢性炎症性疾病、血管钙化、高血压、缺血性心脏病、房颤和中风可能是谷氨酸依赖的症状。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The Glutamate Dependence Hypothesis May Explain Overeating Browned Food Only and the Worldwide Heart Disease Pandemic
The following study tried to determine whether Maillard reaction end product-embodied food is significantly more alluring, rewarding, addictive, and cardiovascular taxing than Maillard reaction end product-free meals. As importantly, does Maillard end product-containing fare exhibit more tolerance measured by increased total calorie consumption per day, progression measured by rising amounts of overweight, and withdrawal measured by more frequent urges to snack and dine than Maillard reaction end product-free provisions? The data presented here suggests an intimate association between dietary Maillard-reaction end product-induced glutamate dependence, overeating browned food only, and cardiovascular disease. Phosphorous and glutamate-prompted redox imbalance, hyperlipidemia, immune system activation, chronic inflammatory disease, vascular calcification, hypertension, ischemic heart disease, atrial fibrillation, and stroke may be symptoms of Glutamate dependence.
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