抑制tlr9依赖性p38 MAPK信号通路可改善NOD/Ltj小鼠原发性Sjögren综合征的发病机制。

IF 0.8 4区 医学 Q4 ENDOCRINOLOGY & METABOLISM
N Cao, H Shi, C Chen, L Zheng, C Yu
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引用次数: 1

摘要

本文章由计算机程序翻译,如有差异,请以英文原文为准。
Inhibition of the TLR9-dependent p38 MAPK signaling pathway improves the pathogenesis of primary Sjögren's syndrome in the NOD/Ltj mouse.
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来源期刊
CiteScore
2.20
自引率
15.60%
发文量
0
审稿时长
6 months
期刊介绍: Journal of Biological Regulators & Homeostatic Agents (IF 1.397) is a peer-reviewed journal published every 2 months. The journal publishes original papers describing research in the fields of experimental and clinical medicine, molecular biology, biochemistry, regulatory molecules, cellular immunology and pharmacology.
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