阻塞性睡眠呼吸暂停综合征中的心律失常:危险并发症还是防御机制?

L S Korostovtseva, S O Kravchenko, Yu V Sviryaev, A O Konradi, G A Oganesyan
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引用次数: 0

摘要

睡眠的特点是不同阶段和阶段的循环和连续交替,每个阶段都有内在的植物调节变化和心率的振荡,特别是在伴有躯体疾病的情况下,可能导致心律障碍的发展。本文讨论了心脏传导障碍与阻塞性睡眠呼吸暂停之间的相互关系问题。研究表明,在睡眠停止状态下发生慢行性心律失常的一些机制(首先是伴随副交感神经张力增加的植物调节特征)是人类潜水员以及在水中或近水生活方式的哺乳动物所固有的,这些哺乳动物在潜水时被迫呼吸延迟和长时间呆在水下。这些机制可能具有防御功能。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[BRADY-ARRHYTHMIAS IN THE OBSTRUCTIVE SLEEP APNEA SYNDROME: DANGEROUS COMPLICATION OR DEFENSE MECHANISM?].

Sleep is characterized by cycling and consecutive alternation of different phases and stages, each of them featuring intrinsic changes of vegetative regulation with oscillations of heartbeat rate that may cause development of cardiac rhythm disorders especially under presence of concomitant somatic diseases. This paper deals with the problems of interrelationships between disturbances of heart conduction and obstruc- tive sleep apnea. It is shown that some mechanisms of the occurrence of brady-arrhythmias (first of all, features of vegetative regulation with increasing parasympathetic tone) under sleep arrests are inherent to human divers as well as to mammals conducting water or near-water way of living associated with forced breathing delays at diving and prolonged staying under water. These mechanisms may carry out defense function.

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