乏力、肌痛和大量CK升高:一种严重的神经系统疾病?

Medizinische Klinik Pub Date : 2010-07-01 Epub Date: 2010-07-30 DOI:10.1007/s00063-010-1084-9
Johannes Steinfurt, Markus C Müller, Anke Seidel, Richard Salm, Andreas Ochs
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引用次数: 1

摘要

病例报告:作者报告了一名51岁的患者,患有短暂性麻痹,肌痛和大量肌酸激酶升高,导致全科医生进行了密集的神经系统检查。尽管难治性高血压,原发性醛固酮增多症不排除。在作者的诊所,病人被诊断出患有康氏综合症。腹腔镜肾上腺切除术发现左侧肾上腺有一个大的腺瘤。结论:高血压患者短暂性神经麻痹、肌痛和肌酸激酶升高可提示原发性醛固酮增多症。如临床怀疑,应测定醛固酮-肾素比值。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Pareses, myalgias, and massive CK elevation: a severe neurological disorder?].

Case report: The authors report on a 51-year-old patient with transient pareses, myalgias, and a massive creatine kinase elevation which had led to an intensive neurological work-up by the general practitioner. Despite refractory hypertension, primary aldosteronism was not excluded. At the authors' clinic, the patient was diagnosed to have Conn's syndrome. Laparoscopic adrenalectomy revealed a big adenoma of the left adrenal gland.

Conclusion: Transient pareses, myalgias, and creatine kinase elevation can indicate primary aldosteronism among hypertensive patients. If clinically suspected, the aldosterone-renin ratio should be determined.

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Medizinische Klinik
Medizinische Klinik 医学-医学:内科
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