实验性旋毛虫病生化防御机制的组织化学研究:1 .过氧化物酶活性。

Tropical medicine and parasitology : official organ of Deutsche Tropenmedizinische Gesellschaft and of Deutsche Gesellschaft fur Technische Zusammenarbeit (GTZ) Pub Date : 1995-12-01

E Hadaás, L Gustowska

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引用次数: 0

摘要

自由基在分子和细胞水平上引起的生物反应涉及许多不同的生化成分，这些生化成分可以被氧化自由基直接破坏，并最终促进病理过程。防御机制已经进化到限制自由基损伤产生的速度;它们包括低分子抗氧化剂和抗氧化酶。本研究分析了旋毛虫感染不同阶段小鼠肌肉中出现过氧化物的过氧化物酶活性。提出的结果是一个更大的研究的一部分，关于宿主在实验旋毛虫病过程中的防御机制。

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Histochemical investigations of the biochemical defence mechanism in experimental trichinellosis: I. peroxidase activity.

Biological reactions caused by free radicals at the molecular and cellular levels engage many different biochemical components which can be directly damaged by the oxidizing radicals and which eventually promote pathological processes. Defence mechanisms have evolved to limit the speed of production of free radical damage; they include low molecular antioxidants and antioxidant enzymes. In this study, the activity of peroxidase acting against peroxides appearing in muscle of mice at different stages of infection with Trichinella spiralis was analyzed. The presented results are part of a bigger research concerning the defence mechanisms of the host in the course of experimental trichinellosis.

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Tropical medicine and parasitology : official organ of Deutsche Tropenmedizinische Gesellschaft and of Deutsche Gesellschaft fur Technische Zusammenarbeit (GTZ)

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