{"title":"心肌梗死和肌肉萎缩症患者血清天冬氨酸转氨酶线粒体同工酶水平的变化。","authors":"T R Boyde","doi":"10.1159/000458267","DOIUrl":null,"url":null,"abstract":"Summary. The cationic (mitochondrial) isoenzyme of aspartate aminotransferase is a normal constituent of plasma. After myocardial infarction the seru~ level usually rises, reaching a peak 24 to 48 h later, and returns to normal more slowly than does the total aspartate aminotransferase activity. The serum level was found to be raised in all 6 cases of muscular dystrophy tested.","PeriodicalId":78243,"journal":{"name":"Enzymologia biologica et clinica","volume":"9 5","pages":"385-92"},"PeriodicalIF":0.0000,"publicationDate":"1968-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000458267","citationCount":"19","resultStr":"{\"title\":\"Serum levels of the mitochondrial isoenzym of aspartate aminotransferase in myocardial infarction and muscular dystrophy.\",\"authors\":\"T R Boyde\",\"doi\":\"10.1159/000458267\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Summary. The cationic (mitochondrial) isoenzyme of aspartate aminotransferase is a normal constituent of plasma. After myocardial infarction the seru~ level usually rises, reaching a peak 24 to 48 h later, and returns to normal more slowly than does the total aspartate aminotransferase activity. The serum level was found to be raised in all 6 cases of muscular dystrophy tested.\",\"PeriodicalId\":78243,\"journal\":{\"name\":\"Enzymologia biologica et clinica\",\"volume\":\"9 5\",\"pages\":\"385-92\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1968-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.1159/000458267\",\"citationCount\":\"19\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Enzymologia biologica et clinica\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1159/000458267\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Enzymologia biologica et clinica","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1159/000458267","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Serum levels of the mitochondrial isoenzym of aspartate aminotransferase in myocardial infarction and muscular dystrophy.
Summary. The cationic (mitochondrial) isoenzyme of aspartate aminotransferase is a normal constituent of plasma. After myocardial infarction the seru~ level usually rises, reaching a peak 24 to 48 h later, and returns to normal more slowly than does the total aspartate aminotransferase activity. The serum level was found to be raised in all 6 cases of muscular dystrophy tested.