肥胖和急性呼吸窘迫综合征中肺泡2型细胞是否“过饱和”？

IF 3.5 2区医学 Q1 PHYSIOLOGY

American journal of physiology. Lung cellular and molecular physiology Pub Date : 2025-10-14 DOI:10.1152/ajplung.00325.2025

Avnee Jaya Kumar, Mark L Hepokoski

引用次数: 0

摘要

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Are alveolar type 2 cells "oversaturated" in obesity and ARDS?

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来源期刊

American journal of physiology. Lung cellular and molecular physiology 医学-呼吸系统

CiteScore

9.20

自引率

4.10%

发文量

146

审稿时长

2 months

期刊介绍： The American Journal of Physiology-Lung Cellular and Molecular Physiology publishes original research covering the broad scope of molecular, cellular, and integrative aspects of normal and abnormal function of cells and components of the respiratory system. Areas of interest include conducting airways, pulmonary circulation, lung endothelial and epithelial cells, the pleura, neuroendocrine and immunologic cells in the lung, neural cells involved in control of breathing, and cells of the diaphragm and thoracic muscles. The processes to be covered in the Journal include gas-exchange, metabolic control at the cellular level, intracellular signaling, gene expression, genomics, macromolecules and their turnover, cell-cell and cell-matrix interactions, cell motility, secretory mechanisms, membrane function, surfactant, matrix components, mucus and lining materials, lung defenses, macrophage function, transport of salt, water and protein, development and differentiation of the respiratory system, and response to the environment.