更正“Tenascin-R通过调节APP/PS1小鼠的Nav1.6活性，加重穿孔通路中Aβ的产生”。

IF 11.1 1区医学 Q1 CLINICAL NEUROLOGY

Alzheimer's & Dementia Pub Date : 2025-09-30 DOI:10.1002/alz.70759

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引用次数: 0

摘要

王斌，王志祥，吕立明，等。Tenascin-R通过调节APP/PS1小鼠的Nav1.6活性，促进穿孔通路中Aβ的产生。老年痴呆症。2025;21(9):e70633。10.1002 / alz。70633文中列出的通讯作者之一为“李少”。正确的名字应该是少里，“少”是名，“里”是姓。我们为这个错误道歉。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

Correction to "Tenascin-R aggravates Aβ production in the perforant pathway by regulating Nav1.6 activity in APP/PS1 mice"

查看原文本刊更多论文

Correction to "Tenascin-R aggravates Aβ production in the perforant pathway by regulating Nav1.6 activity in APP/PS1 mice"

Wang B, Wang ZX, Lv LM, et al. Tenascin-R aggravates Aβ production in the perforant pathway by regulating Nav1.6 activity in APP/PS1 mice. Alzheimers Dement. 2025;21(9):e70633. 10.1002/alz.70633

One of the corresponding authors was listed as “Li Shao” in the article. The correct name should be Shao Li, with “Shao” as the first name and “Li” as the family name.

We apologize for this error.

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来源期刊

Alzheimer's & Dementia 医学-临床神经学

CiteScore

14.50

自引率

5.00%

发文量

299

审稿时长

3 months

期刊介绍： Alzheimer's & Dementia is a peer-reviewed journal that aims to bridge knowledge gaps in dementia research by covering the entire spectrum, from basic science to clinical trials to social and behavioral investigations. It provides a platform for rapid communication of new findings and ideas, optimal translation of research into practical applications, increasing knowledge across diverse disciplines for early detection, diagnosis, and intervention, and identifying promising new research directions. In July 2008, Alzheimer's & Dementia was accepted for indexing by MEDLINE, recognizing its scientific merit and contribution to Alzheimer's research.