从信号到衰老：IFNγ驱动肌炎中的肌肉萎缩

IF 10.6 1区医学 Q1 CLINICAL NEUROLOGY

Brain Pub Date : 2025-06-09 DOI:10.1093/brain/awaf220

Werner Stenzel, Hans-Hilmar Goebel, Marie Holzer

引用次数: 0

摘要

这篇科学评论引用了Hou等人的“干扰素-g导致免疫肌病中的肌源性细胞功能障碍和衰老”（https://doi.org/10.1093/brain/awaf153）。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

查看原文本刊更多论文

From signal to senescence: IFNγ drives muscle atrophy in myositis

This scientific commentary refers to ‘Interferon-g causes myogenic cell dysfunction and senescence in immune myopathies’ by Hou et al. (https://doi.org/10.1093/brain/awaf153).

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来源期刊

Brain 医学-临床神经学

CiteScore

20.30

自引率

4.10%

发文量

458

审稿时长

3-6 weeks

期刊介绍： Brain, a journal focused on clinical neurology and translational neuroscience, has been publishing landmark papers since 1878. The journal aims to expand its scope by including studies that shed light on disease mechanisms and conducting innovative clinical trials for brain disorders. With a wide range of topics covered, the Editorial Board represents the international readership and diverse coverage of the journal. Accepted articles are promptly posted online, typically within a few weeks of acceptance. As of 2022, Brain holds an impressive impact factor of 14.5, according to the Journal Citation Reports.