对“miR-223通过抑制右旋糖酐硫酸钠诱导的实验性结肠炎中IL-6/STAT3信号通路改善肠道炎症”的更正

IF 3.1 4区医学 Q3 IMMUNOLOGY

Immunity, Inflammation and Disease Pub Date : 2025-05-21 DOI:10.1002/iid3.70209

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引用次数: 0

摘要

张军，王超，郭忠，达兵，朱伟，李清，“miR-223通过抑制IL-6/STAT3信号通路改善实验性结肠炎的肠道炎症，”中华免疫杂志，第9期，no。1 (2021): 319-327, https://doi.org/10.1002/iid3.395.The图4D凝胶根据同行评议时审稿人的建议切割。未切割、注释的凝胶已作为补充图提供。作者确认文中提到的所有实验结果和相应结论不受影响。补充图如下图所示。作者对这个错误表示歉意。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Correction to “miR-223 Improves Intestinal Inflammation Through Inhibiting the IL-6/STAT3 Signaling Pathway in Dextran Sodium Sulfate-Induced Experimental Colitis”

J. Zhang, C. Wang, Z. Guo, B. Da, W. Zhu and Q. Li, “miR-223 Improves Intestinal Inflammation Through Inhibiting the IL-6/STAT3 Signaling Pathway in Dextran Sodium Sulfate-Induced Experimental Colitis,” Immunity, Inflammation and Disease 9, no. 1 (2021): 319-327, https://doi.org/10.1002/iid3.395.

The gel in Figure 4D was cut following the reviewer's suggestion during peer review. The uncut, annotated gels have been provided as a supplemental figure. The authors confirm that all the experimental results and corresponding conclusions mentioned in the paper remain unaffected. Supplemental Figure is shown as follows.

Supplemental Figure

The authors apologize for this error.

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来源期刊

Immunity, Inflammation and Disease Medicine-Immunology and Allergy

CiteScore

3.60

自引率

0.00%

发文量

146

审稿时长

8 weeks

期刊介绍： Immunity, Inflammation and Disease is a peer-reviewed, open access, interdisciplinary journal providing rapid publication of research across the broad field of immunology. Immunity, Inflammation and Disease gives rapid consideration to papers in all areas of clinical and basic research. The journal is indexed in Medline and the Science Citation Index Expanded (part of Web of Science), among others. It welcomes original work that enhances the understanding of immunology in areas including: • cellular and molecular immunology • clinical immunology • allergy • immunochemistry • immunogenetics • immune signalling • immune development • imaging • mathematical modelling • autoimmunity • transplantation immunology • cancer immunology