抗淋巴瘤肽的灵感来自于KRAI基序的4个氨基酸序列作为Crlz-1的核定位信号。

Molecular therapy. Oncology Pub Date : 2025-02-20 eCollection Date: 2025-03-20 DOI:10.1016/j.omton.2025.200953
Joo Hyun Pi, Seung Young Choi, Sung-Kyun Park, Junghyun Lim, Chang Joong Kang
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引用次数: 0

摘要

绘制成短 KRAI 序列的 Crlz-1 核定位信号肽抑制了来自伯基特淋巴瘤患者生殖中心的拉莫斯细胞的增殖。这种抗增殖作用的机理是,Crlz-1核移动受阻,CBFβ核动员失败,导致Bcl-6 GC主基因的增强子-启动子上缺乏结合的Runx/CBFβ异源二聚体。由于这种异源二聚体的缺失,Bcl-6 的表达被取消,导致细胞周期蛋白 D1-D3 下调,IRF-4、Blimp-1 和 IgJ 基因上调。此外,该肽还减少了这些细胞中 rRNA 的产生,表明核 Crlz-1 作为核糖体小亚基过程组的 UTP-3 组成成分,可能是调节 rRNA 的生物生成和/或加工所必需的,从而产生了细胞快速增殖所必需的核糖体。令人惊讶的是,KRAI基序肽本身就具有细胞膜渗透性,因此只需将其直接加入培养基或注入尾静脉,就能在培养细胞和拉莫斯异种移植小鼠中显示出抗增殖和抗肿瘤作用。这无疑为开发针对生殖中心源性 B 细胞淋巴瘤的新型抗癌多肽药物铺平了道路。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Anti-lymphoma peptide is inspired by mapping a sequence of four amino acids of KRAI motif as nuclear localization signal of Crlz-1.

Peptides of Crlz-1 nuclear localization signal as mapped to be a short KRAI sequence inhibited the proliferation of germinal center-derived Ramos cells from Burkitt's lymphoma patient. This anti-proliferative effect was mechanistically explained by a cascade of the block of Crlz-1 nuclear movement and consequential failure of CBFβ nuclear mobilization, resulting in the absence of bound Runx/CBFβ heterodimer on the enhancer-promoter of the Bcl-6 GC master gene. As a consequence of this heterodimer absence, the Bcl-6 expression was abolished, leading to the down-regulation of cyclins D1-D3 and the up-regulation of IRF-4, Blimp-1, and IgJ genes. Furthermore, this peptide decreased the production of rRNA in these cells, indicating that the nuclear Crlz-1 as a UTP-3 constituent of ribosomal small subunit processome might be necessary to regulate the biogenesis and/or processing of rRNA, and thereby produce ribosomes necessary for their rapid proliferation. Surprisingly, the KRAI motif peptides had an intrinsic cell-membrane permeability by themselves, and therefore their anti-proliferative and anti-tumor effects were also demonstrated in both the cultured cells and Ramos-xenografted mice just by adding them directly to the culture media or injecting them into tail veins. This definitely paved the prospective road to developing a novel anti-cancer peptide drug against the germinal center-derived B cell lymphoma.

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