CD4+ T细胞重新连接肉芽肿细胞和调节网络，促进Mtb再感染后的免疫调节

IF 25.5 1区医学 Q1 IMMUNOLOGY

Immunity Pub Date : 2025-01-15 DOI:10.1016/j.immuni.2025.01.001

Joshua D. Bromley, Sharie Keanne C. Ganchua, Sarah K. Nyquist, Pauline Maiello, Michael Chao, H. Jacob Borish, Mark Rodgers, Jaime Tomko, Kara Kracinovsky, Douaa Mugahid, Son Nguyen, Qianchang Dennis Wang, Jacob M. Rosenberg, Edwin C. Klein, Hannah P. Gideon, Roisin Floyd-O’Sullivan, Bonnie Berger, Charles A. Scanga, Philana Ling Lin, Sarah M. Fortune, JoAnne L. Flynn

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引用次数: 0

摘要

(免疫57,2380-2398.e1-e6；2024年10月8日)

本文章由计算机程序翻译，如有差异，请以英文原文为准。

查看原文本刊更多论文

CD4+ T cells re-wire granuloma cellularity and regulatory networks to promote immunomodulation following Mtb reinfection

(Immunity 57, 2380–2398.e1–e6; October 8, 2024)

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来源期刊

Immunity 医学-免疫学

CiteScore

49.40

自引率

2.20%

发文量

205

审稿时长

6 months

期刊介绍： Immunity is a publication that focuses on publishing significant advancements in research related to immunology. We encourage the submission of studies that offer groundbreaking immunological discoveries, whether at the molecular, cellular, or whole organism level. Topics of interest encompass a wide range, such as cancer, infectious diseases, neuroimmunology, autoimmune diseases, allergies, mucosal immunity, metabolic diseases, and homeostasis.