{"title":"修正“肝脏信号换能器和转录- 3信号激活因子通过抑制生酮驱动早期胰腺癌恶病质”","authors":"","doi":"10.1002/jcsm.13687","DOIUrl":null,"url":null,"abstract":"<p>Arneson-Wissink P. C., Mendez H., Pelz K., Dickie J., Bartlett A. Q., Worley B. L., et al (2024) Hepatic signal transducer and activator of transcription-3 signalling drives early-stage pancreatic cancer cachexia via suppressed ketogenesis, <i>Journal of Cachexia, Sarcopenia and Muscle</i>. https://doi.org/10.1002/jcsm.13466.</p><p>In our attribution of funding, we mistakenly omitted the following: PCAW was supported by the National Cancer Institute K99CA286709–01.</p><p>We apologize for this error.</p>","PeriodicalId":48911,"journal":{"name":"Journal of Cachexia Sarcopenia and Muscle","volume":"16 1","pages":""},"PeriodicalIF":9.4000,"publicationDate":"2024-12-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcsm.13687","citationCount":"0","resultStr":"{\"title\":\"Correction to ‘Hepatic signal transducer and activator of transcription-3 signalling drives early-stage pancreatic cancer cachexia via suppressed ketogenesis’\",\"authors\":\"\",\"doi\":\"10.1002/jcsm.13687\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Arneson-Wissink P. C., Mendez H., Pelz K., Dickie J., Bartlett A. Q., Worley B. L., et al (2024) Hepatic signal transducer and activator of transcription-3 signalling drives early-stage pancreatic cancer cachexia via suppressed ketogenesis, <i>Journal of Cachexia, Sarcopenia and Muscle</i>. https://doi.org/10.1002/jcsm.13466.</p><p>In our attribution of funding, we mistakenly omitted the following: PCAW was supported by the National Cancer Institute K99CA286709–01.</p><p>We apologize for this error.</p>\",\"PeriodicalId\":48911,\"journal\":{\"name\":\"Journal of Cachexia Sarcopenia and Muscle\",\"volume\":\"16 1\",\"pages\":\"\"},\"PeriodicalIF\":9.4000,\"publicationDate\":\"2024-12-26\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcsm.13687\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Cachexia Sarcopenia and Muscle\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/jcsm.13687\",\"RegionNum\":1,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"GERIATRICS & GERONTOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cachexia Sarcopenia and Muscle","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcsm.13687","RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"GERIATRICS & GERONTOLOGY","Score":null,"Total":0}
Correction to ‘Hepatic signal transducer and activator of transcription-3 signalling drives early-stage pancreatic cancer cachexia via suppressed ketogenesis’
Arneson-Wissink P. C., Mendez H., Pelz K., Dickie J., Bartlett A. Q., Worley B. L., et al (2024) Hepatic signal transducer and activator of transcription-3 signalling drives early-stage pancreatic cancer cachexia via suppressed ketogenesis, Journal of Cachexia, Sarcopenia and Muscle. https://doi.org/10.1002/jcsm.13466.
In our attribution of funding, we mistakenly omitted the following: PCAW was supported by the National Cancer Institute K99CA286709–01.
期刊介绍:
The Journal of Cachexia, Sarcopenia and Muscle is a peer-reviewed international journal dedicated to publishing materials related to cachexia and sarcopenia, as well as body composition and its physiological and pathophysiological changes across the lifespan and in response to various illnesses from all fields of life sciences. The journal aims to provide a reliable resource for professionals interested in related research or involved in the clinical care of affected patients, such as those suffering from AIDS, cancer, chronic heart failure, chronic lung disease, liver cirrhosis, chronic kidney failure, rheumatoid arthritis, or sepsis.