“CD180结扎通过STAT3调节髓源性抑制细胞的积累和免疫抑制活性，从而导致内毒性休克。”生物化学生物物理学报(BBA) - Mol Basis Dis 2019；1865(3): 535 - 546。]。

IF 4.2 2区生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY

Biochimica et biophysica acta. Molecular basis of disease Pub Date : 2024-12-20 DOI:10.1016/j.bbadis.2024.167622

Guanjun Dong , Xiaoying Yao , Fenglian Yan , Hui Zhang , Yuzhen Zhu , Yonghong Yang , Hui Shi , Junfeng Zhang , Zhaochen Ning , Cuiling Wang , Panpan Cheng , Yuan Hu , Qun Ma , Jun Dai , Zhihua Li , Chunxia Li , Jiankuo Ming , Xuehui Li , Chuanping Si , Huabao Xiong

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Corrigendum to “Ligation of CD180 contributes to endotoxic shock by regulating the accumulation and immunosuppressive activity of myeloid-derived suppressor cells through STAT3.” [Biochim Biophys Acta (BBA) - Mol Basis Dis 2019; 1865(3):535–546.]

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来源期刊

Biochimica et biophysica acta. Molecular basis of disease 生物-生化与分子生物学

CiteScore

12.30

自引率

0.00%

发文量

218

审稿时长

32 days

期刊介绍： BBA Molecular Basis of Disease addresses the biochemistry and molecular genetics of disease processes and models of human disease. This journal covers aspects of aging, cancer, metabolic-, neurological-, and immunological-based disease. Manuscripts focused on using animal models to elucidate biochemical and mechanistic insight in each of these conditions, are particularly encouraged. Manuscripts should emphasize the underlying mechanisms of disease pathways and provide novel contributions to the understanding and/or treatment of these disorders. Highly descriptive and method development submissions may be declined without full review. The submission of uninvited reviews to BBA - Molecular Basis of Disease is strongly discouraged, and any such uninvited review should be accompanied by a coverletter outlining the compelling reasons why the review should be considered.