“抑制自噬通过调节ATG5和ATG7降低氟化物诱导的LS8细胞凋亡率”的勘误。

IF 3.2 3区 医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

摘要

赵丽丽,王红华,刘顺生,等,“通过调节ATG5和ATG7抑制自噬降低氟化物诱导的LS8细胞凋亡率,”生物化学与分子毒理学杂志37,no。3(2023年3月):e23280。图2CNaF 24 h组在整理实验结果时,错误复制了NaF 24 h组的错误图像,导致出现错误。我们现在已经更正了,并附上了准确的图片如下。三个实验重复的原始数据已提交给该杂志。图4CAa bcl-2波段在整理实验结果时,错误复制了错误的bcl-2波段图像,导致出现错误。我们现在已经更正了,并附上了准确的图片如下。三个实验重复的原始数据已提交给该杂志。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Erratum to “Inhibition of Autophagy Reduces the Rate of Fluoride-Induced LS8 Apoptosis via Regulating ATG5 and ATG7”

L. Zhao, H. Wang, S. Liu, et al., “Inhibition of Autophagy Reduces the Rate of Fluoride-Induced LS8 Apoptosis via Regulating ATG5 and ATG7,” Journal of Biochemical and Molecular Toxicology 37, no. 3 (March 2023): e23280.

Results:

Figure 2C

NaF 24 h group

While organizing the experimental results, the wrong image of NaF 24 h group was mistakenly copied, leading to an error. We have now corrected it and attached the accurate image below. The raw data of the three experimental replicates was submitted to the journal.

Figure 4C

Aa bcl-2 band

While organizing the experimental results, the wrong image of bcl-2 band was mistakenly copied, leading to an error. We have now corrected it and attached the accurate image below. The raw data of the three experimental replicates was submitted to the journal.

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来源期刊
CiteScore
5.80
自引率
2.80%
发文量
277
审稿时长
6-12 weeks
期刊介绍: The Journal of Biochemical and Molecular Toxicology is an international journal that contains original research papers, rapid communications, mini-reviews, and book reviews, all focusing on the molecular mechanisms of action and detoxication of exogenous and endogenous chemicals and toxic agents. The scope includes effects on the organism at all stages of development, on organ systems, tissues, and cells as well as on enzymes, receptors, hormones, and genes. The biochemical and molecular aspects of uptake, transport, storage, excretion, lactivation and detoxication of drugs, agricultural, industrial and environmental chemicals, natural products and food additives are all subjects suitable for publication. Of particular interest are aspects of molecular biology related to biochemical toxicology. These include studies of the expression of genes related to detoxication and activation enzymes, toxicants with modes of action involving effects on nucleic acids, gene expression and protein synthesis, and the toxicity of products derived from biotechnology.
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