自噬作用有助于达沙替尼诱导的人类急性髓性白血病细胞髓系分化》[Biochem. Pharmacol. 89/1 (2014) 74-85]的更正。

IF 5.3 2区医学 Q1 PHARMACOLOGY & PHARMACY

Biochemical pharmacology Pub Date : 2024-10-01 DOI:10.1016/j.bcp.2024.116553

Nan Xie , Like Zhong , Lu Liu, Yanfeng Fang, Xiaotian Qi, Ji Cao, Bo Yang, Qiaojun He, Meidan Ying

引用次数: 0

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本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Corrigendum to “Autophagy contributes to dasatinib-induced myeloid differentiation of human acute myeloid leukemia cells” [Biochem. Pharmacol. 89/1 (2014) 74–85]

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来源期刊

Biochemical pharmacology 医学-药学

CiteScore

10.30

自引率

1.70%

发文量

420

审稿时长

17 days

期刊介绍： Biochemical Pharmacology publishes original research findings, Commentaries and review articles related to the elucidation of cellular and tissue function(s) at the biochemical and molecular levels, the modification of cellular phenotype(s) by genetic, transcriptional/translational or drug/compound-induced modifications, as well as the pharmacodynamics and pharmacokinetics of xenobiotics and drugs, the latter including both small molecules and biologics. The journal''s target audience includes scientists engaged in the identification and study of the mechanisms of action of xenobiotics, biologics and drugs and in the drug discovery and development process. All areas of cellular biology and cellular, tissue/organ and whole animal pharmacology fall within the scope of the journal. Drug classes covered include anti-infectives, anti-inflammatory agents, chemotherapeutics, cardiovascular, endocrinological, immunological, metabolic, neurological and psychiatric drugs, as well as research on drug metabolism and kinetics. While medicinal chemistry is a topic of complimentary interest, manuscripts in this area must contain sufficient biological data to characterize pharmacologically the compounds reported. Submissions describing work focused predominately on chemical synthesis and molecular modeling will not be considered for review. While particular emphasis is placed on reporting the results of molecular and biochemical studies, research involving the use of tissue and animal models of human pathophysiology and toxicology is of interest to the extent that it helps define drug mechanisms of action, safety and efficacy.