定量蛋白质组学发现阿尔茨海默病中结构蛋白、突触蛋白和记忆相关蛋白的 O-GlcNAcylation 发生了改变 "一文的更正。

IF 5.6 2区医学 Q1 ONCOLOGY

The Journal of Pathology Pub Date : 2024-07-30 DOI:10.1002/path.6342

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引用次数: 0

摘要

Sheng Wang, Feng Yang, Vladislav A Petyuk, Anil K Shukla, Matthew E Monroe, Marina A Gritsenko, Karin D Rodland, Richard D Smith, Wei-Jun Qian, Cheng-Xin Gong and Tao Liu.J Pathol 2017; 243: 78-88. https://doi.org/10.1002/path.4929Tao 刘涛是共同通讯作者之一，他已告知编辑本文补充材料图S1中的一处错误，该文于2017年6月28日首次发表在Wiley Online Library (https://onlinelibrary.wiley.com/)上。另一位共同通讯作者龚承信同意这一更正，两人都对这一错误和造成的不便表示歉意。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

Correction to ‘Quantitative proteomics identifies altered O-GlcNAcylation of structural, synaptic and memory-associated proteins in Alzheimer's disease’

查看原文本刊更多论文

Correction to ‘Quantitative proteomics identifies altered O-GlcNAcylation of structural, synaptic and memory-associated proteins in Alzheimer's disease’

Sheng Wang, Feng Yang, Vladislav A Petyuk, Anil K Shukla, Matthew E Monroe, Marina A Gritsenko, Karin D Rodland, Richard D Smith, Wei-Jun Qian, Cheng-Xin Gong and Tao Liu. J Pathol 2017; 243: 78–88. https://doi.org/10.1002/path.4929

Tao Liu, one of the co-corresponding authors, has informed the editors of an error in supplementary material, Figure S1 of this article, first published on 28 June 2017 in Wiley Online Library (https://onlinelibrary.wiley.com/).

Cheng-Xin Gong, the other co-corresponding author, agrees with this correction, and both apologise for the error and the inconvenience caused.

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来源期刊

The Journal of Pathology 医学-病理学

CiteScore

14.10

自引率

1.40%

发文量

144

审稿时长

3-8 weeks

期刊介绍： The Journal of Pathology aims to serve as a translational bridge between basic biomedical science and clinical medicine with particular emphasis on, but not restricted to, tissue based studies. The main interests of the Journal lie in publishing studies that further our understanding the pathophysiological and pathogenetic mechanisms of human disease. The Journal of Pathology welcomes investigative studies on human tissues, in vitro and in vivo experimental studies, and investigations based on animal models with a clear relevance to human disease, including transgenic systems. As well as original research papers, the Journal seeks to provide rapid publication in a variety of other formats, including editorials, review articles, commentaries and perspectives and other features, both contributed and solicited.