回收。

IF 4.5 2区 生物学 Q2 CELL BIOLOGY
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引用次数: 0

摘要

撤回:S. Mukherjee, S. Manna, D. Pal, P. Mukherjee, C. K. Panda, Sequential loss of cell cycle checkpoint control contributes to malignant transformation of murine embryonic fibroblasts induced by 20-methylcholanthrene, Journal of Cellular Physiology 224, no:49-58, https://doi.org/10.1002/jcp.22089.上述文章于 2010 年 4 月 23 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经期刊主编 Alexander Hutchison 和 Wiley Periodicals LLC 协议,该文章已被撤回。第三方对上述文章中的图片篡改和复制提出了质疑。出版商的调查证实了以下与图像相关的问题:图 3B(i)中 Western 印迹的重复和拼接;图 4 中细胞切片的旋转、大小调整和重复;图 4 和图 6 C 之间细胞切片的重复;图 5B 中 Western 印迹的重复;图 6 A 中 Western 印迹的重复;以及图 6 C 中细胞切片的重复。由于篡改图像的证据非常广泛,而且影响了多张图,期刊认定这些结果从根本上受到了损害,因此必须撤稿。作者不同意撤稿。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION.

Retraction: S. Mukherjee, S. Manna, D. Pal, P. Mukherjee, C. K. Panda, Sequential loss of cell cycle checkpoint control contributes to malignant transformation of murine embryonic fibroblasts induced by 20-methylcholanthrene, Journal of Cellular Physiology 224, no. 1 (2010): 49-58, https://doi.org/10.1002/jcp.22089. The above article, published online on 23 April 2010 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between journal Editor in Chief, Alexander Hutchison; and Wiley Periodicals LLC. Concerns were raised by a third party regarding image manipulation and duplication in the above article. An investigation by the publisher has confirmed the following image-related concerns: duplication and splicing of western blots in Figure 3B(i); rotation, resizing, and duplication of cellular sections in Figure 4; duplication of cellular sections between Figure 4 and Figure 6 C; duplication of western blots in Figure 5B; duplication of western blots in Figure 6 A; and duplication of cellular sections in Figure 6 C. Because the evidence of image manipulation is extensive and impacts several figures, the journal has determined that the results are fundamentally compromised, and so the journal must retract the article. The authors disagree with the retraction.

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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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