抑制 GBP5 可激活自噬,减轻 LPS 诱导的小鼠肺损伤的炎症反应

IF 1.5 4区 医学 Q3 RESPIRATORY SYSTEM
Jialin Li, Kexuan Liu, Wenjuan He, Wencai Zhang, Yongchao Li
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引用次数: 0

摘要

肺气肿是一种长期导致肺组织损伤的疾病。作为鸟苷酸结合蛋白家族的一部分,GBP5 在小鼠肺气肿中调节失调。然而,...
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Inhibition of GBP5 activates autophagy to alleviate inflammatory response in LPS-induced lung injury in mice
Pulmonary emphysema is a condition that causes damage to the lung tissue over time. GBP5, as part of the guanylate-binding protein family, is dysregulated in mouse pulmonary emphysema. However, the...
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来源期刊
Experimental Lung Research
Experimental Lung Research 医学-呼吸系统
CiteScore
3.80
自引率
0.00%
发文量
23
审稿时长
2 months
期刊介绍: Experimental Lung Research publishes original articles in all fields of respiratory tract anatomy, biology, developmental biology, toxicology, and pathology. Emphasis is placed on investigations concerned with molecular, biochemical, and cellular mechanisms of normal function, pathogenesis, and responses to injury. The journal publishes reports on important methodological advances on new experimental modes. Also published are invited reviews on important and timely research advances, as well as proceedings of specialized symposia. Authors can choose to publish gold open access in this journal.
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