[结扎勒死肺充血的机制(四)]。

Igaku kenkyu. Acta medica Pub Date : 1989-03-01
T Kita, Y Furuya
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引用次数: 0

摘要

采用放射免疫法、分光光度法和纤维蛋白板法测定了结扎勒死豚鼠血浆和血清血管舒缩素(肾素、缓激肽、纤溶蛋白、乳酸和铁蛋白)水平的变化。本实验是结扎勒死致肺充血机制研究的一部分。1)与预杀组和击打枕部组相比,结扎勒死组血浆肾素水平显著升高。2)与预杀组相比,结扎勒死组和击打枕部组血浆乳酸水平均显著升高。然而,在上述两组之间,该物质的含量没有显著差异。3)结扎勒死组与预割组血浆缓激肽、纤溶蛋白及血清铁蛋白水平差异无统计学意义。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Mechanisms of the pulmonary congestion in ligature strangulation (IV)].

The authors determined by radioimmunoassay, spectrophotometry or fibrin plate method the changes of levels of plasma or serum vasomotor (renin, bradykinin, plasmin, lactic acid and ferritin) of guinea-pigs sacrificed by ligature strangulation. This experiment was a part of the studies on the mechanisms of the pulmonary congestion in ligature strangulation. 1) In the ligature strangulation group, significant high levels of plasma renin was observed when compared with the presacrificed group and the beating on occiput group. 2) In both the ligature strangulation group and the beating on occiput group, significant high levels of plasma lactic acid were observed when compared with the presacrificed group. However, between the above-mentioned two groups, there were no significant differences in the levels of this substance. 3) Between the ligature strangulation group and the presacrificed group, there were no significant differences in the levels of plasma bradykinin and plasmin and serum ferritin.

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