{"title":"表观遗传异常是致癌扩增的唯一驱动因素","authors":"M Nezami","doi":"10.47363/pms/2023(7)192","DOIUrl":null,"url":null,"abstract":"Three genetic mechanisms, including mutations, gene amplification, and chromosome rearrangements, have been recognized as drivers of oncogene activation in human neoplasms.","PeriodicalId":491076,"journal":{"name":"Progress in medical sciences","volume":"102 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2023-10-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Epigenetic Aberrancies as the Exclusive Driver of Oncogenic Amplifications\",\"authors\":\"M Nezami\",\"doi\":\"10.47363/pms/2023(7)192\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Three genetic mechanisms, including mutations, gene amplification, and chromosome rearrangements, have been recognized as drivers of oncogene activation in human neoplasms.\",\"PeriodicalId\":491076,\"journal\":{\"name\":\"Progress in medical sciences\",\"volume\":\"102 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2023-10-31\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Progress in medical sciences\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.47363/pms/2023(7)192\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Progress in medical sciences","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.47363/pms/2023(7)192","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Epigenetic Aberrancies as the Exclusive Driver of Oncogenic Amplifications
Three genetic mechanisms, including mutations, gene amplification, and chromosome rearrangements, have been recognized as drivers of oncogene activation in human neoplasms.
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