Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway
AMA Gao Y, Liu J, Wang X, Meng Q. Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway. Folia Neuropathologica. 2023. doi:10.5114/fn.2023.131191. APA Gao, Y., Liu, J., Wang, X., & Meng, Q. (2023). Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway. Folia Neuropathologica. https://doi.org/10.5114/fn.2023.131191 Chicago Gao, Yuan, Jiang Liu, Xiaoyan Wang, and Qiang Meng. 2023. "Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway". Folia Neuropathologica. doi:10.5114/fn.2023.131191. Harvard Gao, Y., Liu, J., Wang, X., and Meng, Q. (2023). Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway. Folia Neuropathologica. https://doi.org/10.5114/fn.2023.131191 MLA Gao, Yuan et al. "Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway." Folia Neuropathologica, 2023. doi:10.5114/fn.2023.131191. Vancouver Gao Y, Liu J, Wang X, Meng Q. Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway. Folia Neuropathologica. 2023. doi:10.5114/fn.2023.131191.
期刊介绍:
Folia Neuropathologica is an official journal of the Mossakowski Medical Research Centre Polish Academy of Sciences and the Polish Association of Neuropathologists. The journal publishes original articles and reviews that deal with all aspects of clinical and experimental neuropathology and related fields of neuroscience research. The scope of journal includes surgical and experimental pathomorphology, ultrastructure, immunohistochemistry, biochemistry and molecular biology of the nervous tissue. Papers on surgical neuropathology and neuroimaging are also welcome. The reports in other fields relevant to the understanding of human neuropathology might be considered.