尾侧牵引作为I型Chiari畸形的发病机制

M. B. Royo-Salvador, Marco V. Fiallos-Rivera, H. Salca
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引用次数: 1

摘要

尽管我们对其临床和图像特征的理解取得了重要的成就,但Chiari畸形的发病机制仍然是一个谜,而大多数消除它的努力都是孤立的尝试,处理相当晚的继发性发病事件,如后窝体积缩小,其内容物拥挤或脑脊液在大孔水平的流动紊乱。然而,在新的研究将揭示这些过程的许多方面之前,如果一个人将尾侧牵引理论作为指导原则,那么实际的部分的、碎片化的知识可以以一种更可靠的方式构建。我们提出了一个潜在的发病机制,可能最终导致整个脊髓的异常轴向张力,以及在我们的临床实践中发现的一些图像和治疗特征,证明了这种无情的尾侧牵引。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Caudal Traction as a Pathogenetic Mechanism of Chiari Malformation Type I
Despite the important achievements made with respect to our understanding of their clinical and image features, Chiari malformations are the result of etiopathogenetic mechanisms still sunk into mystery, while most of the efforts to dissipate it are isolated attempts that deal with rather late, secondary pathogenetic events, such as the reduction of the posterior fossa volume, the crowdedness of its contents or the disturbances of the cerebrospinal fluid flow at the level of the foramen magnum. Nevertheless, until new research will shed light onto many of these processes, the actual partial, fragmented knowledge can be structured in a much more reliable manner if one holds the theory of caudal traction as a guiding principle. We present a potential pathogenesis that could culminate into an abnormal axial tension throughout the spinal cord, as well as some image and therapeutic features found during our clinical practice, testifying in favor of this relentless caudal traction.
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