[皮肤受体对反复热刺激反应的变化]。

Neirofiziologiia = Neurophysiology Pub Date : 1992-01-01
A V Zeveke, E D Efes, A S Babikova
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引用次数: 0

摘要

众所周知,皮肤的冷却会引起胶原纤维的收缩,从而导致机械感受器的变形和兴奋。中热作用于皮肤抑制或终止其滋补活动。在这种情况下,胶原纤维伸展,皮肤张力减弱。然而,高达48摄氏度的强烈热刺激会导致更大的传入活动。这种效应与所谓的热感受器有关。前面已经表明,反复的高温刺激会增加皮肤对这种刺激的敏感性。这篇论文报告了记录在温度高达48摄氏度的皮肤反复刺激下传入活动增加的时间的结果,同时记录了皮肤张力的变化。对于每次加热实例,当皮肤温度接近正常温度(即30摄氏度)时,皮肤会拉伸和收缩。皮肤张力未能完全恢复。多次加热试验表明皮肤敏感性下降,传入活动增加的时间缩短。这两个过程是同时发生的,因此可以断定它们是相互关联的。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Changes in the skin receptor responses to their repeated heat stimulation].

It is known that cooling of skin induces contraction of collagen fibres, which leads to deformation and excitation of mechanoreceptors. The action of moderate heat on the skin depresses or terminates its tonic activity. The collagen fibres in this case stretch out and the skin tension slackens. Yet, intense thermal stimulation up to 48 degrees C results in a greater afferent activity. This effect is associated with the so-called thermoreceptors. It is shown earlier that repeated stimulation by intense heat increases skin sensitivity to this stimulus. The paper reports the results of recording the time in which afferent activity increased under repeated stimulation of the skin at temperatures up to 48 degrees C and, simultaneously, of the skin tension variations. For each heating instance the skin stretched and contracted when its temperature approached the normal temperature, i. e. 30 degrees C. However, by the next heating (there was a 3--min. pause between them) the skin tension failed to restore completely. Multiple heating tests have shown a loss of the skin sensitivity, and the time in which increased afferent activity manifested itself has shortened. These two processes are simultaneous, which permits concluding that they are related.

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