低镁日粮对大鼠心肌组织及某些酶活性的影响。

J Wutzen
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摘要

低镁饮食14周的大鼠出现了越来越多的临床症状,从轻微的营养变化到生长抑制。心肌纤维局灶性断裂,继发局灶性嗜紫红细胞增多,随后出现局灶性坏死。形态学病变的增加与钙盐沉积在退化的心肌纤维、结缔组织和心脏血管壁有关。低镁饮食不影响atp酶和细胞色素氧化酶的活性。心肌退行性变和坏死区碱性磷酸酶和琥珀酸脱氢酶无活性。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Effect of low-magnesium diet on the histology and the activity of certain enzymes of rat myocardium.

Rats kept on a low-magnesium diet for 14 weeks developed increasing clinical symptoms, from slight trophic changes to inhibition of growth. Focal fragmentation of myocardial fibers was followed by focal fuchsinophilia and later--by focal necrosis. Increasing morphological lesions were associated with calcium salts deposition in the degenerated myocardial fibers, connective tissue and wall of cardiac blood vessels. The low-magnesium diet did not affect the activity of ATP-ases and cytochrome oxidase. The areas of myocardial degeneration and necrosis were free of the activity of alkaline phosphatase and succinic dehydrogenase.

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